Lopes A T, Manso C
Acta Med Port. 1989 Jan-Feb;2(1):35-9.
The authors review the mechanisms of paraquat and diquat toxicity. They discuss the generation of multiple toxic active forms of oxygen. The pulmonary concentration of paraquat seems to be due to a mechanism of active concentration, which seems to be used by polyamines and diamines in normal situations. Considerations on therapeutics attempts are made.
作者回顾了百草枯和敌草快的毒性机制。他们讨论了多种有毒活性氧形式的产生。百草枯在肺部的浓度似乎归因于一种主动浓缩机制，在正常情况下多胺和二胺似乎会利用这种机制。文中还对治疗尝试进行了探讨。
