Aydin Unal, Yazici Pinar, Alper Isik, Kaplan Hasan
Ege University, Faculty of Medicine, Department of General Surgery, Izmir, Turkey.
Ege University, Faculty of Medicine, Department of Anesthesiology, Izmir, Turkey.
Eurasian J Med. 2009 Apr;41(1):66-9.
In addition to hemorrhage, which is one of the most well-known factors, there are many other causative factors for serious hypotension after hepatic trauma. In this report, we present patients with persistent cardiac depression after perihepatic packing due to high grade liver injury and report on treatment modalities, including the early second-look procedure. Three patients with isolated hepatic trauma were included. Two of the patients who underwent perihepatic packing were transferred from outside hospitals, and one patient required repacking due to severe hemorrhage. All patients had grade IV injuries due to blunt (n=2) or penetrating injury (n=1). In the intensive care unit, central venous pressure (8, 12, 13 mmHg) and hematocrit (26, 27, 29%) were in the normal range, but blood pressure (40/60, 50/70, 45/75mmHg) was abnormal despite the use of inotropic support. The three patients underwent an unpacking procedure 8, 10, and 14 hours later, respectively. Inotropic support was not required after postoperative hours 3, 5, and 6, respectively. The management of post reperfusion syndrome due to hepatic trauma can be achieved, but close collaboration between the surgeon and anesthesiologist is absolutely necessary. In the case of resistance cardiac depression in patients with packing, second-look procedures should be performed as early as possible.
除了出血这一最广为人知的因素外,肝外伤后严重低血压还有许多其他致病因素。在本报告中,我们介绍了因严重肝损伤在肝周填塞后出现持续性心脏抑制的患者,并报告了治疗方式,包括早期二次探查手术。纳入了3例单纯性肝外伤患者。接受肝周填塞的2例患者是从外院转来的,1例患者因严重出血需要再次填塞。所有患者均因钝性伤(n = 2)或穿透伤(n = 1)而有IV级损伤。在重症监护病房,中心静脉压(8、12、13 mmHg)和血细胞比容(26%、27%、29%)在正常范围内,但尽管使用了血管活性药物支持,血压(40/60、50/70、45/75 mmHg)仍不正常。这3例患者分别在8、10和14小时后进行了拆除填塞物手术。术后分别在3、5和6小时后不再需要血管活性药物支持。肝外伤后再灌注综合征的处理是可以实现的,但外科医生和麻醉医生之间的密切合作绝对必要。对于填塞患者出现的顽固性心脏抑制,应尽早进行二次探查手术。