Sato Y, Osaku A, Koyama S, Mizukawa S, Onikura S, Yagi H, Suzuki T, Sawada T, Uchiyama T, Kanmatsuse K
Department of Cardiology, Surugadai Nihon University Hospital, Tokyo, Japan.
Jpn Heart J. 1989 Nov;30(6):935-41. doi: 10.1536/ihj.30.935.
Transient atrioventricular (A-V) conduction abnormalities are often experienced in patients with evolving acute viral myocarditis, but persistent complete A-V block requiring permanent cardiac pacing is rare. We describe a case who developed irreversible complete A-V block during the long-term course of Coxsackie B2 myocarditis. The endomyocardial biopsy revealed inflammatory cellular infiltrates and myocyte necrosis. A left ventriculogram and echocardiogram consistently demonstrated an aneurysm in the basal portion of the interventricular septum. It was speculated that the extensive myocardial scar caused by acute myocarditis resulted in the ventricular aneurysm of this particular myocardial region involving the A-V conduction system.
急性病毒性心肌炎进展期患者常出现短暂性房室传导异常,但需要永久性心脏起搏的持续性完全性房室传导阻滞较为罕见。我们描述了1例在柯萨奇B2型心肌炎长期病程中发生不可逆性完全性房室传导阻滞的病例。心内膜心肌活检显示有炎性细胞浸润和心肌细胞坏死。左心室造影和超声心动图均一致显示室间隔基部有一个动脉瘤。据推测,急性心肌炎所致的广泛心肌瘢痕导致了这个特定心肌区域累及房室传导系统的室壁瘤形成。