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Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis.Myc 的失调需要 MondoA/Mlx 进行代谢重编程和肿瘤发生。
Cancer Cell. 2015 Feb 9;27(2):271-85. doi: 10.1016/j.ccell.2014.11.024. Epub 2015 Jan 29.
2
Interactions between Myc and MondoA transcription factors in metabolism and tumourigenesis.Myc与MondoA转录因子在代谢和肿瘤发生中的相互作用。
Br J Cancer. 2015 Dec 1;113(11):1529-33. doi: 10.1038/bjc.2015.360. Epub 2015 Oct 15.
3
Web of the extended Myc network captures metabolism for tumorigenesis.扩展 Myc 网络的网络捕获了肿瘤发生的代谢。
Cancer Cell. 2015 Feb 9;27(2):160-2. doi: 10.1016/j.ccell.2015.01.004.
4
Coordinated Cross-Talk Between the Myc and Mlx Networks in Liver Regeneration and Neoplasia.Myc 和 Mlx 网络在肝再生和肿瘤发生中的协调对话。
Cell Mol Gastroenterol Hepatol. 2022;13(6):1785-1804. doi: 10.1016/j.jcmgh.2022.02.018. Epub 2022 Mar 5.
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Functional interactions among members of the MAX and MLX transcriptional network during oncogenesis.肿瘤发生过程中MAX和MLX转录网络成员之间的功能相互作用。
Biochim Biophys Acta. 2015 May;1849(5):484-500. doi: 10.1016/j.bbagrm.2014.05.016. Epub 2014 May 22.
6
Metabolic reprogramming in triple-negative breast cancer through Myc suppression of TXNIP.通过Myc抑制TXNIP实现三阴性乳腺癌中的代谢重编程
Proc Natl Acad Sci U S A. 2015 Apr 28;112(17):5425-30. doi: 10.1073/pnas.1501555112. Epub 2015 Apr 13.
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The MNT transcription factor autoregulates its expression and supports proliferation in MYC-associated factor X (MAX)-deficient cells.MTN 转录因子自我调控其表达，并支持 MYC 相关因子 X（MAX）缺陷细胞的增殖。
J Biol Chem. 2020 Feb 14;295(7):2001-2017. doi: 10.1074/jbc.RA119.010389. Epub 2020 Jan 9.
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MondoA, a novel basic helix-loop-helix-leucine zipper transcriptional activator that constitutes a positive branch of a max-like network.MondoA，一种新型的碱性螺旋-环-螺旋-亮氨酸拉链转录激活因子，它构成了一个类Max网络的正性分支。
Mol Cell Biol. 2000 Dec;20(23):8845-54. doi: 10.1128/MCB.20.23.8845-8854.2000.
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c-Myc-driven glycolysis via TXNIP suppression is dependent on glutaminase-MondoA axis in prostate cancer.c-Myc 驱动的糖酵解通过 TXNIP 抑制依赖于前列腺癌中的谷氨酰胺酶-MondoA 轴。
Biochem Biophys Res Commun. 2018 Oct 2;504(2):415-421. doi: 10.1016/j.bbrc.2018.08.069. Epub 2018 Aug 10.
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Glucose controls nuclear accumulation, promoter binding, and transcriptional activity of the MondoA-Mlx heterodimer.葡萄糖控制 MondoA-Mlx 异二聚体的核积累、启动子结合和转录活性。
Mol Cell Biol. 2010 Jun;30(12):2887-95. doi: 10.1128/MCB.01613-09. Epub 2010 Apr 12.
引用本文的文献
1
Hexokinase regulates Mondo-mediated longevity via the PPP and organellar dynamics.己糖激酶通过磷酸戊糖途径（PPP）和细胞器动态变化调节Mondo介导的寿命。
Elife. 2025 Aug 11;12:RP89225. doi: 10.7554/eLife.89225.
2
MAX inactivation deregulates the MYC network and induces neuroendocrine neoplasia in multiple tissues.MAX失活会破坏MYC网络的调控，并在多个组织中诱发神经内分泌肿瘤。
Sci Adv. 2025 Apr 25;11(17):eadt3177. doi: 10.1126/sciadv.adt3177.
3
MLX phosphorylation stabilizes the ChREBP-MLX heterotetramer on tandem E-boxes to control carbohydrate and lipid metabolism.MLX磷酸化可稳定ChREBP-MLX异源四聚体在串联E盒上的结合，从而调控碳水化合物和脂质代谢。
Sci Adv. 2025 Mar 14;11(11):eadt4548. doi: 10.1126/sciadv.adt4548. Epub 2025 Mar 12.
4
A high-throughput screening platform to identify expression inhibitors for liver cancer therapy.一种用于鉴定肝癌治疗表达抑制剂的高通量筛选平台。
Front Oncol. 2025 Feb 14;15:1486671. doi: 10.3389/fonc.2025.1486671. eCollection 2025.
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Oxidative Stress and Reprogramming of Lipid Metabolism in Cancers.癌症中的氧化应激与脂质代谢重编程
Antioxidants (Basel). 2025 Feb 10;14(2):201. doi: 10.3390/antiox14020201.
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The Functional Interaction Between Epstein-Barr Virus and MYC in the Pathogenesis of Burkitt Lymphoma.爱泼斯坦-巴尔病毒与MYC在伯基特淋巴瘤发病机制中的功能相互作用
Cancers (Basel). 2024 Dec 18;16(24):4212. doi: 10.3390/cancers16244212.
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IMPA1-derived inositol maintains stemness in castration-resistant prostate cancer via IMPDH2 activation.IMP A1 衍生的肌醇通过 IMPDH2 的激活来维持去势抵抗性前列腺癌的干性。
J Exp Med. 2024 Nov 4;221(11). doi: 10.1084/jem.20231832. Epub 2024 Oct 29.
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MAX inactivation deregulates the MYC network and induces neuroendocrine neoplasia in multiple tissues.MAX失活会破坏MYC网络，并在多个组织中诱发神经内分泌肿瘤。
bioRxiv. 2024 Sep 24:2024.09.21.614255. doi: 10.1101/2024.09.21.614255.
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Spliceosomal vulnerability of MYCN-amplified neuroblastoma is contingent on PRMT5-mediated regulation of epitranscriptomic and metabolomic pathways.剪接体对 MYCN 扩增神经母细胞瘤的易感性取决于 PRMT5 介导的对表观转录组和代谢组学途径的调节。
Cancer Lett. 2024 Nov 1;604:217263. doi: 10.1016/j.canlet.2024.217263. Epub 2024 Sep 21.
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MLX phosphorylation stabilizes the ChREBP-MLX heterotetramer on tandem E-boxes to control carbohydrate and lipid metabolism.MLX磷酸化可稳定串联E-盒上的ChREBP-MLX异源四聚体，以调控碳水化合物和脂质代谢。
bioRxiv. 2024 Sep 5:2024.09.04.611172. doi: 10.1101/2024.09.04.611172.
本文引用的文献
1
Alteration of the lipid profile in lymphomas induced by MYC overexpression.MYC 过表达诱导的淋巴瘤中脂质谱的改变。
Proc Natl Acad Sci U S A. 2014 Jul 22;111(29):10450-5. doi: 10.1073/pnas.1409778111. Epub 2014 Jul 3.
2
Mitochondrial phosphoenolpyruvate carboxykinase (PEPCK-M) is a pro-survival, endoplasmic reticulum (ER) stress response gene involved in tumor cell adaptation to nutrient availability.线粒体磷酸烯醇式丙酮酸羧激酶（PEPCK-M）是一种促生存的内质网（ER）应激反应基因，参与肿瘤细胞对营养可利用性的适应。
J Biol Chem. 2014 Aug 8;289(32):22090-102. doi: 10.1074/jbc.M114.566927. Epub 2014 Jun 27.
3
Functional interactions among members of the MAX and MLX transcriptional network during oncogenesis.肿瘤发生过程中MAX和MLX转录网络成员之间的功能相互作用。
Biochim Biophys Acta. 2015 May;1849(5):484-500. doi: 10.1016/j.bbagrm.2014.05.016. Epub 2014 May 22.
4
PCK2 activation mediates an adaptive response to glucose depletion in lung cancer.PCK2 激活介导肺癌细胞对葡萄糖耗竭的适应性反应。
Oncogene. 2015 Feb 19;34(8):1044-50. doi: 10.1038/onc.2014.47. Epub 2014 Mar 17.
5
An overview of MYC and its interactome.MYC 及其互作蛋白概述。
Cold Spring Harb Perspect Med. 2014 Jan 1;4(1):a014357. doi: 10.1101/cshperspect.a014357.
6
Sterol regulatory element binding protein-dependent regulation of lipid synthesis supports cell survival and tumor growth.固醇调节元件结合蛋白依赖性的脂质合成调节支持细胞存活和肿瘤生长。
Cancer Metab. 2013 Jan 23;1(1):3. doi: 10.1186/2049-3002-1-3.
7
Cytosolic reducing power preserves glutamate in retina.胞浆还原力可维持视网膜中的谷氨酸。
Proc Natl Acad Sci U S A. 2013 Nov 12;110(46):18501-6. doi: 10.1073/pnas.1311193110. Epub 2013 Oct 14.
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Neuroblastoma and MYCN.神经母细胞瘤与 MYCN
Cold Spring Harb Perspect Med. 2013 Oct 1;3(10):a014415. doi: 10.1101/cshperspect.a014415.
9
Coordination of nutrient availability and utilization by MAX- and MLX-centered transcription networks.由 MAX 和 MLX 为中心的转录网络协调营养物质的可用性和利用。
Cold Spring Harb Perspect Med. 2013 Sep 1;3(9):a014258. doi: 10.1101/cshperspect.a014258.
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Lipogenesis and lipolysis: the pathways exploited by the cancer cells to acquire fatty acids.脂肪生成和脂肪分解：癌细胞获取脂肪酸所利用的途径。
Prog Lipid Res. 2013 Oct;52(4):585-9. doi: 10.1016/j.plipres.2013.08.005. Epub 2013 Aug 31.
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