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Superoxide radical generation by inflamed human synovium after hypoxia.

作者信息

Allen R E, Blake D R, Nazhat N B, Jones P

出版信息

Lancet. 1989 Jul 29;2(8657):282-3. doi: 10.1016/s0140-6736(89)90476-5.

DOI:10.1016/s0140-6736(89)90476-5
PMID:2569096
Abstract
摘要

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Superoxide radical generation by inflamed human synovium after hypoxia.缺氧后炎症状态下的人体滑膜产生超氧阴离子自由基。
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Electron spin resonance spectroscopic demonstration of the generation of reactive oxygen species by diseased human synovial tissue following ex vivo hypoxia-reoxygenation.电子自旋共振光谱法证实离体缺氧-复氧后病变人类滑膜组织产生活性氧。
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Intracellular free radical production in synovial T lymphocytes from patients with rheumatoid arthritis.类风湿关节炎患者滑膜T淋巴细胞内自由基的产生
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引用本文的文献

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The danger model approach to the pathogenesis of the rheumatic diseases.危险模型方法在风湿性疾病发病机制中的应用。
J Immunol Res. 2015;2015:506089. doi: 10.1155/2015/506089. Epub 2015 Apr 20.
2
Hypoxia augments cytokine (transforming growth factor-beta (TGF-beta) and IL-1)-induced vascular endothelial growth factor secretion by human synovial fibroblasts.缺氧增强细胞因子（转化生长因子-β（TGF-β）和白细胞介素-1）诱导人滑膜成纤维细胞分泌血管内皮生长因子的能力。
Clin Exp Immunol. 1999 Jan;115(1):176-82. doi: 10.1046/j.1365-2249.1999.00775.x.
3
Electron spin resonance spectroscopic demonstration of the generation of reactive oxygen species by diseased human synovial tissue following ex vivo hypoxia-reoxygenation.
电子自旋共振光谱法证实离体缺氧-复氧后病变人类滑膜组织产生活性氧。
Ann Rheum Dis. 1995 Feb;54(2):94-9. doi: 10.1136/ard.54.2.94.
4
Oxidative damage to hyaluronate and glucose in synovial fluid during exercise of the inflamed rheumatoid joint. Detection of abnormal low-molecular-mass metabolites by proton-n.m.r. spectroscopy.类风湿性关节炎关节炎症发作时滑液中透明质酸和葡萄糖的氧化损伤。通过质子核磁共振波谱法检测异常低分子量代谢物。
Biochem J. 1991 Jan 15;273(Pt 2)(Pt 2):459-67. doi: 10.1042/bj2730459.
5
Localisation of xanthine oxidase to synovial endothelium.黄嘌呤氧化酶在滑膜内皮细胞中的定位。
Ann Rheum Dis. 1991 Nov;50(11):760-2. doi: 10.1136/ard.50.11.760.
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Comparative study of intra-articular pressure dynamics in joints with acute traumatic and chronic inflammatory effusions: potential implications for hypoxic-reperfusion injury.急性创伤性和慢性炎症性积液关节内压力动态变化的比较研究：对缺氧再灌注损伤的潜在影响
Ann Rheum Dis. 1991 Dec;50(12):917-20. doi: 10.1136/ard.50.12.917.
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Exercise induced release of von Willebrand factor: evidence for hypoxic reperfusion microvascular injury in rheumatoid arthritis.运动诱导的血管性血友病因子释放：类风湿关节炎中缺氧再灌注微血管损伤的证据。
Ann Rheum Dis. 1992 Oct;51(10):1117-22. doi: 10.1136/ard.51.10.1117.
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Stress proteins, self defence, and the myocardium.应激蛋白、自我防御与心肌
Br Heart J. 1992 Apr;67(4):279-80. doi: 10.1136/hrt.67.4.279.