Sernov L N, Snegireva G V, Gatsura V V
Kardiologiia. 1989 Aug;29(8):66-9.
The experiments on rats indicated that fructose-1,6-diphosphate substantially decreased the sizes of a necrotic zone, elevated myocardial ATP levels, reduced the edematization of lung tissue, shortened the duration of early postocclusive arrhythmias, and increased the latent period for their development. A dose-dependent cardioprotective effect was found in the preparation.
对大鼠的实验表明，1,6-二磷酸果糖可显著减小坏死区大小，提高心肌ATP水平，减轻肺组织水肿，缩短闭塞后早期心律失常的持续时间，并延长其发生的潜伏期。该制剂具有剂量依赖性心脏保护作用。
