Pohl Michael B, Kendall Karen D, Patel Chirag, Wiley J Preston, Emery Carolyn, Ferber Reed
Department of Kinesiology and Health Promotion, University of Kentucky, Lexington.
J Athl Train. 2015 Apr;50(4):385-91. doi: 10.4085/1062-6050-49.5.07.
Researchers have postulated that reduced hip-abductor muscle strength may have a role in the progression of knee osteoarthritis by increasing the external knee-adduction moment. However, the relationship between hip-abductor strength and frontal-plane biomechanics remains unclear.
To experimentally reduce hip-abduction strength and observe the subsequent changes in frontal-plane biomechanics.
Descriptive laboratory study.
Research laboratory.
Eight healthy, recreationally active men (age = 27 ± 6 years, height = 1.75 ± 0.11 m, mass = 76.1 ± 10.0 kg).
INTERVENTION(S): All participants underwent a superior gluteal nerve block injection to reduce the force output of the hip-abductor muscle group.
MAIN OUTCOME MEASURE(S): Maximal isometric hip-abduction strength and gait biomechanical data were collected before and after the injections. Gait biomechanical variables collected during walking consisted of knee- and hip-adduction moments and impulses and the peak angles of contralateral pelvic drop, hip adduction, and ipsilateral trunk lean.
Hip-abduction strength was reduced after the injection (P = .001) and remained lower than baseline values at the completion of the postinjection gait data collection (P = .02). No alterations in hip- or knee-adduction moments (hip: P = .11; knee: P = .52) or impulses (hip: P = .16; knee: P = .41) were found after the nerve block. Similarly, no changes in angular kinematics were observed for contralateral pelvic drop (P = .53), ipsilateral trunk lean (P = .78), or hip adduction (P = .48).
A short-term reduction in hip-abductor strength was not associated with alterations in the frontal-plane gait biomechanics of young, healthy men. Further research is needed to determine whether a similar relationship is true in older adults with knee osteoarthritis.
研究人员推测,髋外展肌力量减弱可能通过增加膝关节的外展力矩在膝关节骨关节炎的进展中起作用。然而,髋外展肌力量与额状面生物力学之间的关系仍不清楚。
通过实验降低髋外展肌力量,并观察随后额状面生物力学的变化。
描述性实验室研究。
研究实验室。
8名健康、有休闲运动习惯的男性(年龄=27±6岁,身高=1.75±0.11米,体重=76.1±10.0千克)。
所有参与者均接受了臀上神经阻滞注射,以降低髋外展肌群的力量输出。
在注射前后收集最大等长髋外展肌力量和步态生物力学数据。步行过程中收集的步态生物力学变量包括膝关节和髋关节的内收力矩和冲量,以及对侧骨盆下降、髋关节内收和同侧躯干倾斜的峰值角度。
注射后髋外展肌力量降低(P = 0.001),在注射后步态数据收集完成时仍低于基线值(P = 0.02)。神经阻滞后,髋关节或膝关节的内收力矩(髋关节:P = 0.11;膝关节:P = 0.52)或冲量(髋关节:P = 0.16;膝关节:P = 0.41)均未发现改变。同样,对侧骨盆下降(P = 0.53)、同侧躯干倾斜(P = 0.78)或髋关节内收(P = 0.48)的角运动学也未观察到变化。
年轻健康男性髋外展肌力量的短期降低与额状面步态生物力学的改变无关。需要进一步研究以确定在患有膝关节骨关节炎的老年人中是否也存在类似的关系。