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本文引用的文献

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G protein-linked signaling pathways in bipolar and major depressive disorders.双相和重度抑郁症中的 G 蛋白偶联信号通路。
Front Genet. 2013 Dec 23;4:297. doi: 10.3389/fgene.2013.00297. eCollection 2013.
2
Channelopathy pathogenesis in autism spectrum disorders.自闭症谱系障碍中的通道病发病机制。
Front Genet. 2013 Nov 5;4:222. doi: 10.3389/fgene.2013.00222.
3
Sodium channel SCN8A (Nav1.6): properties and de novo mutations in epileptic encephalopathy and intellectual disability.钠通道SCN8A（Nav1.6）：癫痫性脑病和智力残疾中的特性及新发突变
Front Genet. 2013 Oct 28;4:213. doi: 10.3389/fgene.2013.00213.
4
Autoantibodies to neurotransmitter receptors and ion channels: from neuromuscular to neuropsychiatric disorders.针对神经递质受体和离子通道的自身抗体：从神经肌肉疾病到神经精神疾病
Front Genet. 2013 Sep 20;4:181. doi: 10.3389/fgene.2013.00181.
5
A review of potassium channels in bipolar disorder.双相障碍中钾通道的研究综述。
Front Genet. 2013 Jun 11;4:105. doi: 10.3389/fgene.2013.00105. eCollection 2013.
6
Converging Evidence for Epistasis between ANK3 and Potassium Channel Gene KCNQ2 in Bipolar Disorder.双相障碍中 ANK3 和钾通道基因 KCNQ2 之间的上位性证据汇聚。
Front Genet. 2013 May 17;4:87. doi: 10.3389/fgene.2013.00087. eCollection 2013.
7
Major channels involved in neuropsychiatric disorders and therapeutic perspectives.主要涉及神经精神疾病的通道和治疗视角。
Front Genet. 2013 May 7;4:76. doi: 10.3389/fgene.2013.00076. eCollection 2013.
8
Dysfunction of the Heteromeric KV7.3/KV7.5 Potassium Channel is Associated with Autism Spectrum Disorders.异源 KV7.3/KV7.5 钾通道功能障碍与自闭症谱系障碍有关。
Front Genet. 2013 Apr 16;4:54. doi: 10.3389/fgene.2013.00054. eCollection 2013.
9
Genetics, statistics and human disease: analytical retooling for complexity.遗传学、统计学与人类疾病：针对复杂性的分析工具重塑。
Trends Genet. 2004 Dec;20(12):640-7. doi: 10.1016/j.tig.2004.09.007.

Editorial: "Ion channels and mental illness: exploring etiology and pathophysiology in major psychiatric disorders".

作者信息

Askland Kathleen D

机构信息

Psychiatry, Research and Academics, Waypoint Centre for Mental Health Care/University of Toronto Penetanguishene, ON, Canada.

出版信息

Front Genet. 2015 Apr 24;6:152. doi: 10.3389/fgene.2015.00152. eCollection 2015.

DOI:10.3389/fgene.2015.00152
PMID:25964797
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4408861/
Abstract
摘要