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Myocardial Infarct Scar: Hunting Down the Responsible Cells, But Then What?

作者信息

Monsanto Megan, Sussman Mark A

机构信息

Department of Biology, San Diego State University Heart Institute, San Diego State University, San Diego, California.

Department of Biology, San Diego State University Heart Institute, San Diego State University, San Diego, California.

出版信息

J Am Coll Cardiol. 2015 May 19;65(19):2067-9. doi: 10.1016/j.jacc.2015.03.519.

DOI:10.1016/j.jacc.2015.03.519
PMID:25975468
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6703819/
Abstract
摘要

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本文引用的文献

1
Interacting resident epicardium-derived fibroblasts and recruited bone marrow cells form myocardial infarction scar.心肌梗死后瘢痕由相互作用的心肌内源性成纤维细胞和募集的骨髓细胞形成。
J Am Coll Cardiol. 2015 May 19;65(19):2057-66. doi: 10.1016/j.jacc.2015.03.520.
2
Resident fibroblast lineages mediate pressure overload-induced cardiac fibrosis.驻留成纤维细胞谱系介导压力超负荷诱导的心脏纤维化。
J Clin Invest. 2014 Jul;124(7):2921-34. doi: 10.1172/JCI74783. Epub 2014 Jun 17.
3
Essentials of recombinase-based genetic fate mapping in mice.小鼠中基于重组酶的遗传命运图谱要点
Methods Mol Biol. 2014;1092:437-54. doi: 10.1007/978-1-60327-292-6_26.
4
Origin, development, and differentiation of cardiac fibroblasts.心脏成纤维细胞的起源、发展和分化。
J Mol Cell Cardiol. 2014 May;70:2-8. doi: 10.1016/j.yjmcc.2013.11.003. Epub 2013 Nov 11.
5
WT1 regulates epicardial epithelial to mesenchymal transition through β-catenin and retinoic acid signaling pathways.WT1 通过 β-连环蛋白和视黄酸信号通路调节心外膜上皮向间充质转化。
Dev Biol. 2011 Aug 15;356(2):421-31. doi: 10.1016/j.ydbio.2011.05.668. Epub 2011 May 30.
6
Origins of cardiac fibroblasts.心脏成纤维细胞的起源。
Circ Res. 2010 Nov 26;107(11):1304-12. doi: 10.1161/CIRCRESAHA.110.231910.
7
Potential therapeutic targets for cardiac fibrosis: TGFbeta, angiotensin, endothelin, CCN2, and PDGF, partners in fibroblast activation.心脏纤维化的潜在治疗靶点:TGFβ、血管紧张素、内皮素、CCN2 和 PDGF,成纤维细胞激活的伙伴。
Circ Res. 2010 Jun 11;106(11):1675-80. doi: 10.1161/CIRCRESAHA.110.217737.
8
Cardiac fibroblast: the renaissance cell.心脏成纤维细胞:复兴之细胞。
Circ Res. 2009 Dec 4;105(12):1164-76. doi: 10.1161/CIRCRESAHA.109.209809.
9
Pericytes. Morphofunction, interactions and pathology in a quiescent and activated mesenchymal cell niche.周细胞。静止和激活的间充质细胞微环境中的形态功能、相互作用及病理学
Histol Histopathol. 2009 Jul;24(7):909-69. doi: 10.14670/HH-24.909.
10
Regulation of matrix metalloproteinases by cytokines and reactive oxygen/nitrogen species in the myocardium.细胞因子和活性氧/氮物种对心肌中基质金属蛋白酶的调控
Heart Fail Rev. 2004 Jan;9(1):43-51. doi: 10.1023/B:HREV.0000011393.40674.13.