Argon: a novel therapeutic option to treat neuronal ischemia and reperfusion injuries?
作者信息
Ulbrich Felix, Goebel Ulrich
机构信息
Department of Anesthesiology and Intensive Care Medicine, University Medical Center, Freiburg, Germany.
出版信息
Neural Regen Res. 2015 Jul;10(7):1043-4. doi: 10.4103/1673-5374.160071.
Abstract
摘要
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本文引用的文献
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Argon inhalation attenuates retinal apoptosis after ischemia/reperfusion injury in a time- and dose-dependent manner in rats.氩气吸入以时间和剂量依赖性方式减轻大鼠缺血/再灌注损伤后的视网膜细胞凋亡。
PLoS One. 2014 Dec 23;9(12):e115984. doi: 10.1371/journal.pone.0115984. eCollection 2014.
2
Dose dependent neuroprotection of the noble gas argon after cardiac arrest in rats is not mediated by K(ATP)-channel opening.在大鼠心脏骤停后，惰性气体氩的剂量依赖性神经保护作用不是通过 K(ATP)通道开放介导的。
Resuscitation. 2014 Jun;85(6):826-32. doi: 10.1016/j.resuscitation.2014.02.014. Epub 2014 Feb 28.
3
Postresuscitation treatment with argon improves early neurological recovery in a porcine model of cardiac arrest.氩气复苏后治疗可改善猪心搏骤停模型的早期神经功能恢复。
Shock. 2014 Jan;41(1):72-8. doi: 10.1097/SHK.0000000000000049.
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Argon reduces neurohistopathological damage and preserves functional recovery after cardiac arrest in rats.氩气可减轻心跳骤停后大鼠的神经组织病理损伤，并保护其功能恢复。
Br J Anaesth. 2013 Jun;110 Suppl 1:i106-12. doi: 10.1093/bja/aes509. Epub 2013 Feb 7.
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Ex vivo and in vivo neuroprotection induced by argon when given after an excitotoxic or ischemic insult.兴奋性毒性或缺血性损伤后给予氩气诱导的离体和体内神经保护作用。
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