Smith Gregory T, Davis Heather A
Department of Psychology, University of Kentucky, Lexington, KY, USA.
J Child Psychol Psychiatry. 2015 Nov;56(11):1165-7. doi: 10.1111/jcpp.12455.
A number of recent advances in eating disorders research have helped clarify the nature of risk for the development of such disorders. Culbert et al. () provide an empirical and thoughtful review of these recent advances. The authors identified empirically established risk factors in each of several categories of risk for eating disorders: genetic influences, neurotransmitter activity, hormones, personality, and sociocultural influences. We highlight three implications of their review. First, the review can serve as an important asset to eating disorder researchers, both substantively, by providing a comprehensive account of empirically supported risk processes; and methodologically, by highlighting good standards of evidence for acceptance of a candidate risk factor. Second, eating disorder risk is increased by both transdiagnostic and eating disorder-specific factors; there is a need to understand how these types of factors transact with each other. Third and most important, we highlight the importance of Culbert et al.'s advocacy for the development of theoretical models, and empirical tests of those models that specify transactions among different types of risk factors, such as those based on genetic, neurobiological, personality, and social processes.
饮食失调研究领域最近取得的一些进展有助于阐明此类失调症发展的风险本质。卡尔伯特等人()对这些最新进展进行了实证且深入的综述。作者们确定了饮食失调几类风险中每一类经实证确立的风险因素:基因影响、神经递质活动、激素、人格以及社会文化影响。我们强调他们综述的三点启示。首先,该综述对饮食失调症研究人员而言可成为一项重要资产,从实质上来说,它全面阐述了经实证支持的风险过程;从方法上来说,它突出了接受候选风险因素的良好证据标准。其次,跨诊断因素和特定于饮食失调症的因素都会增加饮食失调风险;有必要了解这些因素类型之间是如何相互作用的。第三也是最重要的一点,我们强调卡尔伯特等人倡导建立理论模型以及对那些明确不同类型风险因素(如基于基因、神经生物学、人格和社会过程的因素)之间相互作用的模型进行实证检验的重要性。