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The effect of rhododendrol inhibition of NF-κB on melanocytes in the presence of tyrosinase.

作者信息

Arase Noriko, Yang Lingli, Tanemura Atsushi, Yang Fei, Suenaga Tadahiro, Arase Hisashi, Katayama Ichiro

机构信息

Department of Dermatology, Osaka University Graduate School of Medicine, Suita, Osaka, Japan; Department of Immunochemistry, Research Institute for Microbial Diseases, Osaka University, Suita, Osaka, Japan.

Department of Dermatology, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

出版信息

J Dermatol Sci. 2016 Aug;83(2):157-9. doi: 10.1016/j.jdermsci.2016.05.002. Epub 2016 May 3.

DOI:10.1016/j.jdermsci.2016.05.002
PMID:27174091
Abstract
摘要

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引用本文的文献

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Rhododendrol-induced leukoderma update II: Pathophysiology, mechanisms, risk evaluation, and possible mechanism-based treatments in comparison with vitiligo.杜鹃醇诱导的白癜风研究进展II:病理生理学、发病机制、风险评估以及与白癜风相比基于可能机制的治疗方法
J Dermatol. 2021 Jul;48(7):969-978. doi: 10.1111/1346-8138.15878. Epub 2021 May 5.
2
Biochemical Mechanism of Rhododendrol-Induced Leukoderma.杜鹃醇诱导白癜风的生化机制。
Int J Mol Sci. 2018 Feb 12;19(2):552. doi: 10.3390/ijms19020552.
3
Loss of TAB3 expression by shRNA exhibits suppressive bioactivity and increased chemical sensitivity of ovarian cancer cell lines via the NF-κB pathway.
通过短发夹RNA(shRNA)使TAB3表达缺失,可通过核因子κB(NF-κB)途径表现出抑制性生物活性并增强卵巢癌细胞系的化学敏感性。
Cell Prolif. 2016 Dec;49(6):657-668. doi: 10.1111/cpr.12293. Epub 2016 Sep 21.