Golloub C A, Marcelo N C, Leahy W
Department of Pediatrics, University of Medicine and Dentistry of New Jersey, Newark 07103.
South Med J. 1989 May;82(5):650-2. doi: 10.1097/00007611-198905000-00029.
In the case we have reported, there is a combination of factors that can be attributed to SLE and its associated complications, which may explain the transient hypodensities seen on cranial CT scan. Vasculitis and uncontrolled hypertension lead to arterial spasm, ischemia, and increased vascular permeability, all of which eventually cause cerebral edema. This edema may manifest itself clinically as profound but transient neurologic deficits.
在我们报道的病例中，存在多种可归因于系统性红斑狼疮（SLE）及其相关并发症的因素，这可能解释了头颅CT扫描上所见的短暂性低密度影。血管炎和未控制的高血压导致动脉痉挛、缺血以及血管通透性增加，所有这些最终都会引起脑水肿。这种脑水肿在临床上可能表现为严重但短暂的神经功能缺损。
