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完全性心脏传导阻滞合并登革出血热

Complete Heart Block in Association with Dengue Hemorrhagic Fever.

作者信息

Virk Hafeez Ul Hassan, Inayat Faisal, Rahman Zia Ur

机构信息

Department of Medicine, Mount Sinai St. Luke's and Mount Sinai Roosevelt Hospitals, Icahn School of Medicine, New York, NY, USA.

Department of Medicine, New York-Presbyterian Hospital, Weill Cornell Medical College, New York, NY, USA.

出版信息

Korean Circ J. 2016 Nov;46(6):866-869. doi: 10.4070/kcj.2016.46.6.866. Epub 2016 Oct 13.

DOI:10.4070/kcj.2016.46.6.866
PMID:27826348
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5099345/
Abstract

Dengue virus infection affects the heart structurally and functionally. Clinical manifestations of cardiac complications secondary to dengue virus infection vary from self-limiting arrhythmias to severe myocardial infarction, leading to hypotension, pulmonary edema, and cardiogenic shock. However, we report a case of dengue hemorrhagic fever (DHF) complicated by a complete heart block. A female with DHF due to dengue virus serotype 2, presented to the emergency department with fever, headache, rash, and fatigue followed by an episode of syncope. She was found to have a third-degree atrioventricular block, with pulseless polymorphic ventricular tachycardia. Patient was resuscitated and a temporary trans-venous pacemaker was placed. She reverted back to normal sinus rhythm after 4 days of syncope and was subsequently discharged from the hospital after complete resolution of symptoms, without the need for a permanent pacemaker. Physicians are warranted to have high index of suspicion for dengue virus infection as an etiology in patients with acute cardiovascular compromise, especially in tropical areas.

摘要

登革病毒感染会在结构和功能上影响心脏。登革病毒感染继发的心脏并发症的临床表现各不相同,从自限性心律失常到严重心肌梗死,可导致低血压、肺水肿和心源性休克。然而,我们报告了一例登革出血热(DHF)并发完全性心脏传导阻滞的病例。一名因2型登革病毒感染导致DHF的女性患者,因发热、头痛、皮疹和疲劳就诊于急诊科,随后出现晕厥。她被发现患有三度房室传导阻滞,并伴有无脉性多形性室性心动过速。患者经复苏后植入了临时经静脉起搏器。晕厥4天后她恢复为正常窦性心律,症状完全缓解后出院,无需植入永久性起搏器。对于急性心血管功能不全的患者,尤其是在热带地区,医生有必要高度怀疑登革病毒感染是病因。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3ede/5099345/19323c283005/kcj-46-866-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3ede/5099345/80ecab329d2d/kcj-46-866-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3ede/5099345/128b03cc77b4/kcj-46-866-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3ede/5099345/8e71a670e12e/kcj-46-866-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3ede/5099345/19323c283005/kcj-46-866-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3ede/5099345/80ecab329d2d/kcj-46-866-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3ede/5099345/128b03cc77b4/kcj-46-866-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3ede/5099345/8e71a670e12e/kcj-46-866-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3ede/5099345/19323c283005/kcj-46-866-g004.jpg

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