Sławuta Agnieszka, Kliś Magdalena, Skoczyński Przemysław, Bańkowski Tomasz, Moszczyńska-Stulin Joanna, Gajek Jacek
Department of Cardiology, Klodzko County Hospital, Kłodzko, Poland.
Department of Cardiology, Wroclaw Provincial Specialist Hospital, Poland.
Adv Clin Exp Med. 2016 Sep-Oct;25(5):845-850. doi: 10.17219/acem/63351.
Patients treated for sick sinus syndrome may have interatrial conduction disorder leading to atrial fibrillation.
This study was aimed to assess the influence of the atrial pacing site on interatrial and atrioventricular conduction as well as the percentage of ventricular pacing in patients with sick sinus syndrome implanted with atrioventricular pacemaker.
The study population: 96 patients (58 females, 38 males) aged 74.1 ± 11.8 years were divided in two groups: Group 1 (n = 44) with right atrial appendage pacing and group 2 (n = 52) with Bachmann's area pacing. We assessed the differences in atrioventricular conduction in sinus rhythm and atrial 60 and 90 bpm pacing, P-wave duration and percentage of ventricular pacing.
No differences in baseline P-wave duration in sinus rhythm between the groups (102.4 ± 17 ms vs. 104.1 ± 26 ms, p = ns.) were noted. Atrial pacing 60 bpm resulted in longer P-wave in group 1 vs. group 2 (138.3 ± 21 vs. 106.1 ± 15 ms, p < 0.01). The differences between atrioventricular conduction time during sinus rhythm and atrial pacing at 60 and 90 bpm were significantly longer in patients with right atrial appendage vs. Bachmann's pacing (44.1 ± 17 vs. 9.2 ± 7 ms p < 0.01 and 69.2 ± 31 vs. 21.4 ± 12 ms p < 0.05, respectively). The percentage of ventricular pacing was higher in group 1 (21 vs. 4%, p < 0.01).
Bachmann's bundle pacing decreases interatrial and atrioventricular conduction delay. Moreover, the frequency-dependent atrioventricular conduction lengthening is much less pronounced during Bachmann's bundle pacing. Right atrial appendage pacing in sick sinus syndrome patients promotes a higher percentage of ventricular pacing.
病态窦房结综合征患者接受治疗时可能发生房间传导障碍,进而导致心房颤动。
本研究旨在评估房室起搏器植入的病态窦房结综合征患者中,心房起搏部位对房间及房室传导以及心室起搏百分比的影响。
研究对象:96例患者(58例女性,38例男性),年龄74.1±11.8岁,分为两组:1组(n = 44)采用右心耳起搏,2组(n = 52)采用巴赫曼束区起搏。我们评估了窦性心律、心房60次/分和90次/分起搏时房室传导、P波时限及心室起搏百分比的差异。
两组窦性心律时的基线P波时限无差异(102.4±17毫秒对104.1±26毫秒,p =无统计学意义)。心房60次/分起搏时,1组的P波长于2组(138.3±21对106.1±15毫秒,p < 0.01)。右心耳起搏患者与巴赫曼束区起搏患者相比,窦性心律时及心房60次/分和90次/分起搏时的房室传导时间差异显著更长(分别为44.1±17对9.2±7毫秒,p < 0.01;69.2±31对21.4±12毫秒,p < 0.05)。1组的心室起搏百分比更高(21%对4%,p < 0.01)。
巴赫曼束起搏可减少房间和房室传导延迟。此外,在巴赫曼束起搏期间,频率依赖性房室传导延长不太明显。病态窦房结综合征患者右心耳起搏会导致更高的心室起搏百分比。