Sasaji Tatsuro, Imaizumi Hideki, Takano Hiroyuki, Saitoh Hideo, Murakami Taishi, Kanabuchi Ryuichi, Sekiya Motohiko
Department of Orthopedic Surgery, Osaki Citizen Hospital, 3-8-1 Furukawa Honami, Osaki-shi, Miyagi 989-6183, Japan.
Sekiya Orthopedic Surgery, 5-1-19 Furukawa Ekimae Odori, Osaki-shi, Miyagi 989-6182, Japan.
Case Rep Med. 2016;2016:9412315. doi: 10.1155/2016/9412315. Epub 2016 Dec 6.
While 22 articles have reported on sacral stress fractures, it is a rare injury and its etiology is not well known. We present the case of a 16-year-old male who presented with low back pain in 2015. He was a high school soccer player with a previous history of a bilateral L5 lumbar spondylolysis in 2014. The patient refrained from soccer and wore a brace for six months. Two months after restarting soccer, he again complained of low back pain. After 1 year, a lumbar spine computed tomography revealed the bone union of the spondylolysis. At his first visit to our hospital, his general and neurological conditions were normal and laboratory data were within the normal range. Sacral coronal magnetic resonance imaging (MRI) of the left sacral ala revealed an oblique lineal signal void surrounding bone marrow edema. Based on his symptoms, sports history, and MRI, he was diagnosed with a sacral stress fracture. He again refrained from soccer; his low back pain soon improved, and, after 1 year, the abnormal signal change had disappeared on sacral MRI. Recurrent low back pain case caused by a sacral stress fracture occurring after the bone union of lumbar spondylolysis is uncommon.
虽然已有22篇文章报道了骶骨应力性骨折，但这是一种罕见的损伤，其病因尚不清楚。我们报告一例16岁男性病例，该患者于2015年出现腰痛。他是一名高中足球运动员，2014年曾有双侧L5腰椎峡部裂病史。患者停止踢足球并佩戴支具六个月。重新开始踢足球两个月后，他再次抱怨腰痛。1年后，腰椎计算机断层扫描显示峡部裂骨愈合。在他首次就诊于我院时，其一般情况和神经系统状况正常，实验室数据在正常范围内。左侧骶骨翼的骶骨冠状面磁共振成像（MRI）显示围绕骨髓水肿的斜行线状信号缺失。根据他的症状、运动史和MRI检查，他被诊断为骶骨应力性骨折。他再次停止踢足球；他的腰痛很快改善，1年后，骶骨MRI上的异常信号改变消失。腰椎峡部裂骨愈合后发生骶骨应力性骨折导致的复发性腰痛病例并不常见。
