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Diet-induced dysmotility and neuropathy in the gut precedes endotoxaemia and metabolic syndrome: the chicken and the egg revisited.饮食诱导的肠道运动障碍和神经病变先于内毒素血症和代谢综合征：重新审视因果关系。
J Physiol. 2017 Mar 1;595(5):1441-1442. doi: 10.1113/JP273888. Epub 2017 Feb 13.
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Metabolic endotoxaemia: is it more than just a gut feeling?代谢性内毒素血症：这不仅仅是一种感觉？
Curr Opin Lipidol. 2013 Feb;24(1):78-85. doi: 10.1097/MOL.0b013e32835b4431.
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Apple-Derived Pectin Modulates Gut Microbiota, Improves Gut Barrier Function, and Attenuates Metabolic Endotoxemia in Rats with Diet-Induced Obesity.苹果来源的果胶可调节肠道微生物群，改善肠道屏障功能，并减轻饮食诱导肥胖大鼠的代谢性内毒素血症。
Nutrients. 2016 Feb 29;8(3):126. doi: 10.3390/nu8030126.
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Selective increases of bifidobacteria in gut microflora improve high-fat-diet-induced diabetes in mice through a mechanism associated with endotoxaemia.肠道微生物群中双歧杆菌的选择性增加通过与内毒素血症相关的机制改善高脂饮食诱导的小鼠糖尿病。
Diabetologia. 2007 Nov;50(11):2374-83. doi: 10.1007/s00125-007-0791-0. Epub 2007 Sep 6.
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Changes in gut microbiota control metabolic endotoxemia-induced inflammation in high-fat diet-induced obesity and diabetes in mice.肠道微生物群的变化控制高脂饮食诱导的小鼠肥胖和糖尿病中代谢性内毒素血症引发的炎症。
Diabetes. 2008 Jun;57(6):1470-81. doi: 10.2337/db07-1403. Epub 2008 Feb 27.
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A model of metabolic syndrome and related diseases with intestinal endotoxemia in rats fed a high fat and high sucrose diet.高脂高糖饮食喂养大鼠的代谢综合征及相关疾病伴肠道内毒素血症模型。
PLoS One. 2014 Dec 11;9(12):e115148. doi: 10.1371/journal.pone.0115148. eCollection 2014.
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Protective effect of quercetin on high-fat diet-induced non-alcoholic fatty liver disease in mice is mediated by modulating intestinal microbiota imbalance and related gut-liver axis activation.槲皮素对高脂饮食诱导的小鼠非酒精性脂肪性肝病的保护作用是通过调节肠道微生物群失衡和相关肠-肝轴激活来介导的。
Free Radic Biol Med. 2017 Jan;102:188-202. doi: 10.1016/j.freeradbiomed.2016.11.037. Epub 2016 Nov 25.
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Pepsin egg white hydrolysate ameliorates metabolic syndrome in high-fat/high-dextrose fed rats.胃蛋白酶卵清白水解物可改善高脂肪/高葡萄糖喂养大鼠的代谢综合征。
Food Funct. 2018 Jan 24;9(1):78-86. doi: 10.1039/c7fo01280b.
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Influence of a high-fat diet on gut microbiota, intestinal permeability and metabolic endotoxaemia.高脂肪饮食对肠道微生物群、肠道通透性和代谢内毒素血症的影响。
Br J Nutr. 2012 Sep;108(5):801-9. doi: 10.1017/S0007114512001213. Epub 2012 Apr 16.
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Impact of dietary fat on gut microbiota and low-grade systemic inflammation: mechanisms and clinical implications on obesity.膳食脂肪对肠道微生物群和低度全身炎症的影响：肥胖的机制及临床意义。
Int J Food Sci Nutr. 2018 Mar;69(2):125-143. doi: 10.1080/09637486.2017.1343286. Epub 2017 Jul 4.
引用本文的文献
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High-fat diet-induced alterations to gut microbiota and gut-derived lipoteichoic acid contributes to the development of enteric neuropathy.高脂肪饮食诱导的肠道微生物群和肠道衍生的脂磷壁酸的改变导致肠神经病变的发生。
Neurogastroenterol Motil. 2020 Jul;32(7):e13838. doi: 10.1111/nmo.13838. Epub 2020 Mar 13.
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Intestinal nerve cell injury occurs prior to insulin resistance in female mice ingesting a high-fat diet.高脂饮食摄入的雌性小鼠在发生胰岛素抵抗之前就出现了肠道神经细胞损伤。
Cell Tissue Res. 2019 Jun;376(3):325-340. doi: 10.1007/s00441-019-03002-0. Epub 2019 Feb 18.
本文引用的文献
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Western diet induces colonic nitrergic myenteric neuropathy and dysmotility in mice via saturated fatty acid- and lipopolysaccharide-induced TLR4 signalling.西方饮食通过饱和脂肪酸和脂多糖诱导的Toll样受体4（TLR4）信号传导，诱发小鼠结肠含氮能的肌间神经病变和运动障碍。
J Physiol. 2017 Mar 1;595(5):1831-1846. doi: 10.1113/JP273269. Epub 2017 Feb 8.
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Impact of the gut microbiota on inflammation, obesity, and metabolic disease.肠道微生物群对炎症、肥胖和代谢性疾病的影响。
Genome Med. 2016 Apr 20;8(1):42. doi: 10.1186/s13073-016-0303-2.
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Diabetic gastrointestinal motility disorders and the role of enteric nervous system: current status and future directions.糖尿病性胃肠动力障碍与肠神经系统的作用：现状与未来方向
Neurogastroenterol Motil. 2014 May;26(5):611-24. doi: 10.1111/nmo.12330. Epub 2014 Mar 24.
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Complex interactions among diet, gastrointestinal transit, and gut microbiota in humanized mice.在人源化小鼠中，饮食、胃肠道转运和肠道微生物群之间的复杂相互作用。
Gastroenterology. 2013 May;144(5):967-77. doi: 10.1053/j.gastro.2013.01.047. Epub 2013 Feb 1.
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TLR4 and Insulin Resistance.TLR4 与胰岛素抵抗。
Gastroenterol Res Pract. 2010;2010. doi: 10.1155/2010/212563. Epub 2010 Aug 10.
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