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本文引用的文献

1
Zika virus produces noncoding RNAs using a multi-pseudoknot structure that confounds a cellular exonuclease.
Science. 2016 Dec 2;354(6316):1148-1152. doi: 10.1126/science.aah3963. Epub 2016 Nov 10.
2
Full Genome Sequence and sfRNA Interferon Antagonist Activity of Zika Virus from Recife, Brazil.
PLoS Negl Trop Dis. 2016 Oct 5;10(10):e0005048. doi: 10.1371/journal.pntd.0005048. eCollection 2016 Oct.
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Zika virus - reigniting the TORCH.
Nat Rev Microbiol. 2016 Nov;14(11):707-715. doi: 10.1038/nrmicro.2016.125. Epub 2016 Aug 30.
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New hypotheses derived from the structure of a flaviviral Xrn1-resistant RNA: Conservation, folding, and host adaptation.
RNA Biol. 2015;12(11):1169-77. doi: 10.1080/15476286.2015.1094599. Epub 2015 Sep 23.
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Dengue virus RNA structure specialization facilitates host adaptation.
PLoS Pathog. 2015 Jan 30;11(1):e1004604. doi: 10.1371/journal.ppat.1004604. eCollection 2015 Jan.
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The structural basis of pathogenic subgenomic flavivirus RNA (sfRNA) production.
Science. 2014 Apr 18;344(6181):307-10. doi: 10.1126/science.1250897.
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A highly structured, nuclease-resistant, noncoding RNA produced by flaviviruses is required for pathogenicity.
Cell Host Microbe. 2008 Dec 11;4(6):579-91. doi: 10.1016/j.chom.2008.10.007.
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Viral RNA pseudoknots: versatile motifs in gene expression and replication.
Nat Rev Microbiol. 2007 Aug;5(8):598-610. doi: 10.1038/nrmicro1704.

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