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Variability in local action potential durations, dispersion of repolarization and wavelength restitution in aged wild-type and Scn5a mouse hearts modeling human Brugada syndrome.

作者信息

Tse Gary, Wong Sheung Ting, Tse Vivian, Yeo Jie Ming

机构信息

Department of Medicine and Therapeutics, Faculty of Medicine; Li Ka Shing Institute of Health Sciences, Faculty of Medicine, Chinese University of Hong Kong, Hong Kong, China.

Faculty of Medicine, Imperial College London, London, UK.

出版信息

J Geriatr Cardiol. 2016 Nov;13(11):930-931. doi: 10.11909/j.issn.1671-5411.2016.11.009.

DOI:10.11909/j.issn.1671-5411.2016.11.009
PMID:28133471
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5253411/
Abstract
摘要

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本文引用的文献

1
(Tpeak - Tend)/QRS and (Tpeak - Tend)/(QT × QRS): novel markers for predicting arrhythmic risk in the Brugada syndrome.(T峰 - T终末)/QRS及(T峰 - T终末)/(QT×QRS):预测Brugada综合征心律失常风险的新型标志物。
Europace. 2017 Apr 1;19(4):696. doi: 10.1093/europace/euw194.
2
Electrophysiological Mechanisms of Brugada Syndrome: Insights from Pre-clinical and Clinical Studies.布加综合征的电生理机制:临床前和临床研究的见解
Front Physiol. 2016 Oct 18;7:467. doi: 10.3389/fphys.2016.00467. eCollection 2016.
3
Cardiac disease and arrhythmogenesis: Mechanistic insights from mouse models.心脏疾病与心律失常的发生机制:来自小鼠模型的机制性见解
Int J Cardiol Heart Vasc. 2016 Sep;12:1-10. doi: 10.1016/j.ijcha.2016.05.005.
4
Traditional and novel electrocardiographic conduction and repolarization markers of sudden cardiac death.心脏性猝死的传统及新型心电图传导和复极标志物
Europace. 2017 May 1;19(5):712-721. doi: 10.1093/europace/euw280.
5
Novel arrhythmic risk markers incorporating QRS dispersion: QRS × (T - T )/QRS and QRS × (T - T )/(QT × QRS).纳入QRS离散度的新型心律失常风险标志物:QRS×(T - T)/QRS和QRS×(T - T)/(QT×QRS)
Ann Noninvasive Electrocardiol. 2017 Nov;22(6). doi: 10.1111/anec.12397. Epub 2016 Aug 18.
6
Monophasic action potential recordings: which is the recording electrode?单相动作电位记录:哪个是记录电极?
J Basic Clin Physiol Pharmacol. 2016 Sep 1;27(5):457-62. doi: 10.1515/jbcpp-2016-0007.
7
Depolarization vs. repolarization: what is the mechanism of ventricular arrhythmogenesis underlying sodium channel haploinsufficiency in mouse hearts?去极化与复极化:小鼠心脏中钠通道单倍体不足导致室性心律失常发生的机制是什么?
Acta Physiol (Oxf). 2016 Dec;218(4):234-235. doi: 10.1111/apha.12694. Epub 2016 Apr 30.
8
Action potential wavelength restitution predicts alternans and arrhythmia in murine Scn5a(+/-) hearts.动作电位波长恢复预测 Scn5a(+/-) 小鼠心脏的交替和心律失常。
J Physiol. 2013 Sep 1;591(17):4167-88. doi: 10.1113/jphysiol.2013.254938. Epub 2013 Jul 8.
9
Refractory dispersion promotes conduction disturbance and arrhythmias in a Scn5a (+/-) mouse model.难治性弥散促进 Scn5a(+/-) 小鼠模型中的传导障碍和心律失常。
Pflugers Arch. 2011 Oct;462(4):495-504. doi: 10.1007/s00424-011-0989-3. Epub 2011 Jul 21.
10
Spatial and temporal heterogeneities are localized to the right ventricular outflow tract in a heterozygotic Scn5a mouse model.空间和时间异质性定位于杂合 Scn5a 小鼠模型的右心室流出道。
Am J Physiol Heart Circ Physiol. 2011 Feb;300(2):H605-16. doi: 10.1152/ajpheart.00824.2010. Epub 2010 Nov 19.