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进行性大块纤维化形式的矽肺：一项诊断挑战。

Silicosis in the form of progressive massive fibrosis: A diagnostic challenge.

作者信息

Bhattacharya Somnath, Dey Atin, Pal Amitava, Kar Saurav, Saha Sayantan

机构信息

Department of Pulmonary Medicine, R.G. Kar Medical College and Hospital, Kolkata, West Bengal, India.

出版信息

Indian J Occup Environ Med. 2016 May-Aug;20(2):114-117. doi: 10.4103/0019-5278.197548.

DOI:10.4103/0019-5278.197548
PMID:28194086
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5299811/
Abstract

Silicosis is a major occupational lung disease with a relatively fatal and irreversible outcome. Early diagnosis for shifting the potential candidates to safe modes of workplace as well as for prevention of further progression is the cornerstone of management. Here, we present a complicated case of silicosis in the form of progressive massive fibrosis, which was initially interpreted as tuberculosis; radiological images had resemblance with tuberculosis and cryptogenic organizing pneumonia. Radiology-guided trucut biopsy was imperative to confirm the diagnosis.

摘要

矽肺是一种主要的职业性肺病，其后果相对致命且不可逆转。早期诊断对于将潜在患者转移到安全的工作场所模式以及预防疾病进一步发展是管理的基石。在此，我们呈现一例以进行性大块纤维化形式出现的复杂矽肺病例，该病例最初被误诊为肺结核；影像学图像与肺结核和隐源性机化性肺炎相似。放射学引导下的切割活检对于确诊至关重要。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/febb/5299811/ae00b3f556aa/IJOEM-20-114-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/febb/5299811/7018c3903847/IJOEM-20-114-g001.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/febb/5299811/64665a904fc0/IJOEM-20-114-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/febb/5299811/50a7436e275f/IJOEM-20-114-g004.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/febb/5299811/ae00b3f556aa/IJOEM-20-114-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/febb/5299811/7018c3903847/IJOEM-20-114-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/febb/5299811/d2a2ea185e84/IJOEM-20-114-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/febb/5299811/64665a904fc0/IJOEM-20-114-g003.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/febb/5299811/ae00b3f556aa/IJOEM-20-114-g006.jpg

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Lipopolysaccharides promote pulmonary fibrosis in silicosis through the aggravation of apoptosis and inflammation in alveolar macrophages.脂多糖通过加重肺泡巨噬细胞的凋亡和炎症来促进矽肺中的肺纤维化。
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