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本文引用的文献

1
Peptide- and proton-driven allosteric clamps catalyze anthrax toxin translocation across membranes.肽和质子驱动的变构钳催化炭疽毒素跨膜转运。
Proc Natl Acad Sci U S A. 2016 Aug 23;113(34):9611-6. doi: 10.1073/pnas.1600624113. Epub 2016 Aug 9.
2
Ratcheting up protein translocation with anthrax toxin.炭疽毒素促进蛋白易位。
Protein Sci. 2012 May;21(5):606-24. doi: 10.1002/pro.2052. Epub 2012 Mar 30.
3
Blockage of anthrax PA63 pore by a multicharged high-affinity toxin inhibitor.炭疽芽孢杆菌 PA63 孔道被多电荷高亲和力毒素抑制剂阻塞。
Biophys J. 2010 Jul 7;99(1):134-43. doi: 10.1016/j.bpj.2010.03.070.
4
Protein translocation through the anthrax toxin transmembrane pore is driven by a proton gradient.蛋白质通过炭疽毒素跨膜孔的转运由质子梯度驱动。
J Mol Biol. 2006 Feb 3;355(5):968-79. doi: 10.1016/j.jmb.2005.11.030. Epub 2005 Dec 1.
5
A phenylalanine clamp catalyzes protein translocation through the anthrax toxin pore.苯丙氨酸钳催化蛋白质通过炭疽毒素孔道进行转运。
Science. 2005 Jul 29;309(5735):777-81. doi: 10.1126/science.1113380.
6
Interaction of Clostridium perfringens iota-toxin with lipid bilayer membranes. Demonstration of channel formation by the activated binding component Ib and channel block by the enzyme component Ia.产气荚膜梭菌iota毒素与脂质双分子层膜的相互作用。活化的结合成分Ib形成通道及酶成分Ia阻断通道的证明。
J Biol Chem. 2002 Feb 22;277(8):6143-52. doi: 10.1074/jbc.M103939200. Epub 2001 Dec 10.
7
Interaction of Clostridium botulinum C2 toxin with lipid bilayer membranes. Formation of cation-selective channels and inhibition of channel function by chloroquine.肉毒梭菌C2毒素与脂质双分子层膜的相互作用。阳离子选择性通道的形成及氯喹对通道功能的抑制作用。
J Biol Chem. 1994 Jun 17;269(24):16706-11.
8
Voltage-dependent block of anthrax toxin channels in planar phospholipid bilayer membranes by symmetric tetraalkylammonium ions. Single-channel analysis.平面磷脂双分子层膜中对称四烷基铵离子对炭疽毒素通道的电压依赖性阻断。单通道分析。
J Gen Physiol. 1990 Nov;96(5):921-42. doi: 10.1085/jgp.96.5.921.

Relevance of the alternate conductance states of anthrax toxin channel.

作者信息

Yamini Goli, Nestorovich Ekaterina M

机构信息

Department of Biology, The Catholic University of America, Washington, DC 20064.

Department of Biology, The Catholic University of America, Washington, DC 20064

出版信息

Proc Natl Acad Sci U S A. 2017 Mar 28;114(13):E2545-E2546. doi: 10.1073/pnas.1701841114. Epub 2017 Mar 15.

DOI:10.1073/pnas.1701841114
PMID:28298532
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5380022/
Abstract
摘要