Semmel M, Mercier G, Pavloff N, Dambrine G, Gay F, Biquard J M
U.A. 1158 CNRS, Institut G. Roussy, Villejuif, France.
Arch Virol. 1988;100(1-2):121-9. doi: 10.1007/BF01310914.
In cells infected with Vesicular Stomatitis virus (VSV) ts 1026 and superinfected with Rous Sarcoma virus (RSV) synthesis of vsrc mRNA and RSV env mRNA decreases. In these cells post-translational processing of RSV precursor proteins is impaired and small amounts of VSV antigens are detected.
在感染水疱性口炎病毒（VSV）ts 1026并再次感染劳氏肉瘤病毒（RSV）的细胞中，vsrc mRNA和RSV env mRNA的合成减少。在这些细胞中，RSV前体蛋白的翻译后加工受到损害，并且检测到少量VSV抗原。
