Lim Joel Louis, Tan Kimberly-Anne, Hey Hwee Weng Dennis
University Orthopaedics, Hand and Reconstructive Microsurgery Cluster (UOHC), National University Health System, Singapore.
J Spine Surg. 2017 Mar;3(1):76-81. doi: 10.21037/jss.2017.03.03.
This case report describes the first case of a bone bridge formation across the left L5/S1 neuroforamen after instrumented posterolateral fusion for L5/S1 isthmic spondylolisthesis. Our patient was a 70-year-old lady who had grade 2, L5/S1 isthmic spondylolisthesis and bilateral S1 nerve root compression. She suffered from mechanical low back pain and neurogenic claudication, with radicular pain over both S1 dermatomes. She underwent in-situ, instrumented, posterolateral fusion and was asymptomatic for more than 13 years before developing progressive onset of left radicular pain over the L5 dermatome. Imaging revealed a bisected left L5/S1 neuroforamen secondary to a bone bridge formation resulting in stenosis. The pars defect in this case may have had sufficient osteogenic and osteoinductive factors to heal following spinal stabilization. Although in-situ posterolateral fusion is an accepted surgical treatment for isthmic spondylolisthesis, surgeons should consider reduction of the spondylolisthesis and excision of the pars defects to avoid this possible long-term complication.
本病例报告描述了首例在L5/S1峡部裂型腰椎滑脱后路器械融合术后,横跨左侧L5/S1神经孔形成骨桥的病例。我们的患者是一位70岁女性,患有2级L5/S1峡部裂型腰椎滑脱和双侧S1神经根受压。她患有机械性下腰痛和神经源性间歇性跛行,双侧S1皮节有放射性疼痛。她接受了原位器械辅助后外侧融合术,在出现L5皮节逐渐加重的左侧放射性疼痛之前,无症状超过13年。影像学检查显示,左侧L5/S1神经孔因骨桥形成而被一分为二,导致狭窄。该病例中的峡部缺损可能具有足够的成骨和成骨诱导因子,在脊柱稳定后得以愈合。虽然原位后外侧融合是峡部裂型腰椎滑脱公认有效的手术治疗方法,但外科医生应考虑对腰椎滑脱进行复位并切除峡部缺损,以避免这种可能出现的长期并发症。