Aortic Aneurysms.
作者信息
Lu Hong, Daugherty Alan
机构信息
From the Department of Physiology, Saha Cardiovascular Research Center, University of Kentucky, Lexington.
出版信息
Arterioscler Thromb Vasc Biol. 2017 Jun;37(6):e59-e65. doi: 10.1161/ATVBAHA.117.309578.
Abstract
摘要
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Aortic Aneurysms.主动脉瘤
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Monocytes and macrophages in abdominal aortic aneurysm.腹主动脉瘤中的单核细胞和巨噬细胞。
Nat Rev Cardiol. 2017 Aug;14(8):457-471. doi: 10.1038/nrcardio.2017.52. Epub 2017 Apr 13.
2
Glycated Hemoglobin Is Associated With the Growth Rate of Abdominal Aortic Aneurysms: A Substudy From the VIVA (Viborg Vascular) Randomized Screening Trial.糖化血红蛋白与腹主动脉瘤生长速率相关：来自VIVA（维堡血管）随机筛查试验的一项子研究。
Arterioscler Thromb Vasc Biol. 2017 Apr;37(4):730-736. doi: 10.1161/ATVBAHA.116.308874. Epub 2017 Feb 9.
3
NLRP3 (Nucleotide Oligomerization Domain-Like Receptor Family, Pyrin Domain Containing 3)-Caspase-1 Inflammasome Degrades Contractile Proteins: Implications for Aortic Biomechanical Dysfunction and Aneurysm and Dissection Formation.NLRP3（含核苷酸寡聚化结构域样受体家族吡啉结构域3）-半胱天冬酶-1炎性小体降解收缩蛋白：对主动脉生物力学功能障碍及动脉瘤和夹层形成的影响
Arterioscler Thromb Vasc Biol. 2017 Apr;37(4):694-706. doi: 10.1161/ATVBAHA.116.307648. Epub 2017 Feb 2.
4
Transforming Growth Factor-β in Thoracic Aortic Aneurysms: Good, Bad, or Irrelevant?转化生长因子-β在胸主动脉瘤中的作用：有益、有害还是无关？
J Am Heart Assoc. 2017 Jan 24;6(1):e005221. doi: 10.1161/JAHA.116.005221.
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Aortopathy in a Mouse Model of Marfan Syndrome Is Not Mediated by Altered Transforming Growth Factor β Signaling.马凡综合征小鼠模型中的主动脉病变并非由转化生长因子β信号改变介导。
J Am Heart Assoc. 2017 Jan 24;6(1):e004968. doi: 10.1161/JAHA.116.004968.
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Arterioscler Thromb Vasc Biol. 2017 Mar;37(3):401-410. doi: 10.1161/ATVBAHA.116.308534. Epub 2017 Jan 5.
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The Paraoxonase Gene Cluster Protects Against Abdominal Aortic Aneurysm Formation.对氧磷酶基因簇可预防腹主动脉瘤形成。
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Lifetime Risk and Risk Factors for Abdominal Aortic Aneurysm in a 24-Year Prospective Study: The ARIC Study (Atherosclerosis Risk in Communities).一项为期24年的前瞻性研究：社区动脉粥样硬化风险研究（ARIC研究）中腹主动脉瘤的终生风险及风险因素
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Smooth muscle cell-specific Tgfbr1 deficiency promotes aortic aneurysm formation by stimulating multiple signaling events.平滑肌细胞特异性Tgfbr1缺陷通过刺激多种信号事件促进主动脉瘤形成。
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