Tang Y, Boutilier R G
Department of Biology, Dalhousie University, Halifax, Nova Scotia, Canada.
Exp Biol. 1988;48(1):41-4.
Acute lactacidosis was induced by intra-arterial infusion of lactic acid in seawater-adapted (SW) rainbow trout to examine the dynamics of the metabolic H+ load (delta Hm+) and La- load (delta La-) in the blood. The results were compared with an analogous study on freshwater-adapted (FW) trout by Turner et al. (1983). Following lactic acid infusion, SW trout experienced a less severe acid-base disturbance, and recovered much more quickly than did FW trout. The rates of disappearance of delta Hm+ and delta La- from blood in SW trout were unequal, resulting in a H+ deficit (delta La- - delta Hm+, signs considered) not seen in the FW animals. Unlike the findings on FW trout by Turner et al. (1983), the present data indicate that differential removal of H+ from blood, through net transfers to the environmental water, probably plays an important role in the development of the H+ deficit in SW trout.
通过向适应海水（SW）的虹鳟鱼动脉内输注乳酸来诱导急性乳酸性酸中毒，以研究血液中代谢性H⁺负荷（ΔHm⁺）和乳酸负荷（ΔLa⁻）的动态变化。将结果与Turner等人（1983年）对适应淡水（FW）的鳟鱼进行的类似研究进行比较。输注乳酸后，SW鳟鱼经历的酸碱紊乱较轻，并且比FW鳟鱼恢复得快得多。SW鳟鱼血液中ΔHm⁺和ΔLa⁻的消失速率不相等，导致出现了FW动物中未见到的H⁺亏缺（考虑符号为ΔLa⁻ - ΔHm⁺）。与Turner等人（1983年）对FW鳟鱼的研究结果不同，目前的数据表明，通过净转移至环境水中而从血液中差异性去除H⁺，可能在SW鳟鱼H⁺亏缺的形成中起重要作用。
