人乳头瘤病毒2型E7蛋白对人表皮角质形成细胞中核因子κB活性的减弱作用 - Suppr

Attenuated Nuclear Factor Kappa B Activity by E7 Protein of Human Papillomavirus Type 2 in Human Epidermal Keratinocytes.

作者信息

Ryu Yun Hee, Han Ju Hee, Lee Ji Hyun, Choi Soon Young, Park Young Min

机构信息

Department of Dermatology, Seoul St. Mary's Hospital, College of Medicine, The Catholic University of Korea, Seoul, Korea.

Department of Biological Sciences and Biotechnology, Hannam University, Daejeon, Korea.

出版信息

Ann Dermatol. 2017 Jun;29(3):367-370. doi: 10.5021/ad.2017.29.3.367. Epub 2017 May 11.

DOI:10.5021/ad.2017.29.3.367

PMID:28566923

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5438953/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4a49/5438953/9d0a7917d9a3/ad-29-367-g001.jpg

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Attenuated Nuclear Factor Kappa B Activity by E7 Protein of Human Papillomavirus Type 2 in Human Epidermal Keratinocytes.人乳头瘤病毒2型E7蛋白对人表皮角质形成细胞中核因子κB活性的减弱作用

Ann Dermatol. 2017 Jun;29(3):367-370. doi: 10.5021/ad.2017.29.3.367. Epub 2017 May 11.

Casein kinase II activities related to hyperphosphorylation of human papillomavirus type 16-E7 oncoprotein in epidermal keratinocytes.与表皮角质形成细胞中人乳头瘤病毒16型E7癌蛋白过度磷酸化相关的酪蛋白激酶II活性

Biochem Biophys Res Commun. 1990 Oct 30;172(2):958-64. doi: 10.1016/0006-291x(90)90769-j.

Human papillomavirus type 16 E6 and E7 cooperate to increase epidermal growth factor receptor (EGFR) mRNA levels, overcoming mechanisms by which excessive EGFR signaling shortens the life span of normal human keratinocytes.16型人乳头瘤病毒的E6和E7蛋白协同作用，提高表皮生长因子受体（EGFR）的mRNA水平，克服了因EGFR信号过度而缩短正常人角质形成细胞寿命的机制。

Cancer Res. 2001 May 1;61(9):3837-43.

Epidermal growth factor induction of human papillomavirus type 16 E6/E7 MRNA in tumor cells involves two AP-1 binding sites in the viral enhancer.表皮生长因子诱导肿瘤细胞中16型人乳头瘤病毒E6/E7信使核糖核酸的过程涉及病毒增强子中的两个活化蛋白-1结合位点。

J Gen Virol. 1995 Aug;76 ( Pt 8):1945-58. doi: 10.1099/0022-1317-76-8-1945.

Papillomavirus type 16 oncogenes downregulate expression of interferon-responsive genes and upregulate proliferation-associated and NF-kappaB-responsive genes in cervical keratinocytes.16型乳头瘤病毒致癌基因可下调宫颈角质形成细胞中干扰素反应基因的表达，并上调增殖相关基因和NF-κB反应基因的表达。

J Virol. 2001 May;75(9):4283-96. doi: 10.1128/JVI.75.9.4283-4296.2001.

High levels of p105 (NFKB1) and p100 (NFKB2) proteins in HPV16-transformed keratinocytes: role of E6 and E7 oncoproteins.人乳头瘤病毒16型转化的角质形成细胞中高水平的p105（NFKB1）和p100（NFKB2）蛋白：E6和E7癌蛋白的作用

Virology. 2005 Jan 20;331(2):357-66. doi: 10.1016/j.virol.2004.10.030.

Protein profiling and identification of modulators regulated by human papillomavirus 16 E7 oncogene in HaCaT keratinocytes by proteomics.通过蛋白质组学对人乳头瘤病毒16 E7癌基因在HaCaT角质形成细胞中调控的调节剂进行蛋白质谱分析和鉴定。

Gynecol Oncol. 2005 Oct;99(1):142-52. doi: 10.1016/j.ygyno.2005.05.039.

The relative ability of human papillomavirus type 6 and human papillomavirus type 16 E7 proteins to transactivate E2F-responsive elements is promoter- and cell-dependent.人乳头瘤病毒6型和人乳头瘤病毒16型E7蛋白反式激活E2F反应元件的相对能力取决于启动子和细胞。

Virology. 1997 Dec 8;239(1):238-46. doi: 10.1006/viro.1997.8885.

Expression of human papillomavirus type 16 E7 oncoprotein alters keratinocytes expression profile in response to tumor necrosis factor-alpha.人乳头瘤病毒 16 型 E7 癌蛋白的表达改变角质形成细胞对肿瘤坏死因子-α的反应谱。

Carcinogenesis. 2010 Mar;31(3):521-31. doi: 10.1093/carcin/bgp333. Epub 2009 Dec 30.

Tumor necrosis factor-alpha promotes human papillomavirus (HPV) E6/E7 RNA expression and cyclin-dependent kinase activity in HPV-immortalized keratinocytes by a ras-dependent pathway.肿瘤坏死因子-α通过ras依赖途径促进人乳头瘤病毒（HPV）永生化角质形成细胞中HPV E6/E7 RNA的表达及细胞周期蛋白依赖性激酶活性。

Mol Carcinog. 2000 Feb;27(2):97-109. doi: 10.1002/(sici)1098-2744(200002)27:2<97::aid-mc5>3.0.co;2-v.

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Human papillomavirus-related diseases of the female lower genital tract: oncogenic aspects and molecular interaction.女性下生殖道人乳头瘤病毒相关疾病：致癌机制及分子相互作用

Coll Antropol. 2014 Jun;38(2):779-86.

Topical immunotherapy with diphenylcyclopropenone is effective and preferred in the treatment of periungual warts.用二苯环丙烯酮进行局部免疫疗法在治疗甲周疣方面有效且是首选方法。

Ann Dermatol. 2013 Nov;25(4):434-9. doi: 10.5021/ad.2013.25.4.434. Epub 2013 Nov 30.

New alternative combination therapy for recalcitrant common warts: the efficacy of imiquimod 5% cream and duct tape combination therapy.顽固性寻常疣的新型替代联合疗法：5%咪喹莫特乳膏与胶带联合疗法的疗效

Ann Dermatol. 2013 May;25(2):261-3. doi: 10.5021/ad.2013.25.2.261. Epub 2013 May 10.

Epithelial cell responses to infection with human papillomavirus.人乳头瘤病毒感染的上皮细胞反应。

Clin Microbiol Rev. 2012 Apr;25(2):215-22. doi: 10.1128/CMR.05028-11.

Human papillomavirus type 16 E6 and E 7 proteins alter NF-kB in cultured cervical epithelial cells and inhibition of NF-kB promotes cell growth and immortalization.人乳头瘤病毒 16 型 E6 和 E7 蛋白改变宫颈上皮细胞中的 NF-κB，抑制 NF-κB 促进细胞生长和永生化。

Virology. 2012 Mar 30;425(1):53-60. doi: 10.1016/j.virol.2011.12.023. Epub 2012 Jan 28.

Varicella-zoster virus inhibition of the NF-κB pathway during infection of human dendritic cells: role for open reading frame 61 as a modulator of NF-κB activity.水痘带状疱疹病毒在感染人树突状细胞过程中对 NF-κB 通路的抑制：开放阅读框 61 作为 NF-κB 活性调节剂的作用。

J Virol. 2012 Jan;86(2):1193-202. doi: 10.1128/JVI.06400-11. Epub 2011 Nov 16.

Crosstalk in NF-κB signaling pathways.NF-κB 信号通路中的串扰。

Nat Immunol. 2011 Jul 19;12(8):695-708. doi: 10.1038/ni.2065.

Epstein-Barr virus-encoded EBNA1 inhibits the canonical NF-kappaB pathway in carcinoma cells by inhibiting IKK phosphorylation.EB 病毒编码的 EBNA1 通过抑制 IKK 磷酸化抑制癌细胞中的经典 NF-κB 通路。

Mol Cancer. 2010 Jan 5;9:1. doi: 10.1186/1476-4598-9-1.

The human papillomavirus E7 oncoprotein.人乳头瘤病毒E7癌蛋白

Virology. 2009 Feb 20;384(2):335-44. doi: 10.1016/j.virol.2008.10.006. Epub 2008 Nov 12.

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J Korean Med Sci. 2008 Apr;23(2):307-14. doi: 10.3346/jkms.2008.23.2.307.

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Attenuated Nuclear Factor Kappa B Activity by E7 Protein of Human Papillomavirus Type 2 in Human Epidermal Keratinocytes.

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