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[猫“温热性”心动过速和“寒冷性”心动过缓的机制]

[Mechanisms of "warm" tachycardia and "cold" bradycardia in cats].

作者信息

Samonina G E, Abushinova N N

出版信息

Fiziol Zh SSSR Im I M Sechenova. 1988 May;74(5):714-8.

PMID:2901372
Abstract

The infusion of a small volume of blood with temperature higher ("warm") or below ("cold") the temperature of atrial blood into the right atrium induced "warm" tachycardia and "cold" bradycardia, resp., in anesthetized and in alert cats. Infusion of the blood with temperature equal to that of atrial blood induced no changes of the heart rate. Infusion of the same volume of "cold" or "warm" blood after adreno- and cholinoreceptor-blocking agents was accompanied by a less severe tachycardia or bradycardia. There seem to be two mechanisms in the initiation of the above effects: the neural and the myogenic ones.

摘要

向麻醉和清醒的猫的右心房注入少量温度高于(“温热”)或低于(“冷”)心房血液温度的血液,分别诱发“温热”性心动过速和“冷”性心动过缓。注入与心房血液温度相同的血液时,心率无变化。在使用肾上腺素能和胆碱能受体阻滞剂后注入相同体积的“冷”或“温热”血液,伴随的心动过速或心动过缓程度较轻。上述效应的引发似乎有两种机制:神经机制和肌源性机制。

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