Miyazaki S, Itasaka Y, Yamakawa K, Okawa M, Togawa K
Department of Otolaryngology, Akita University School of Medicine, Japan.
Am J Otolaryngol. 1989 Mar-Apr;10(2):143-9. doi: 10.1016/0196-0709(89)90138-5.
The pathophysiologic results of respiratory disturbances caused by adenoid-tonsillar hypertrophy are reported. In each case, we monitored intraesophageal pressure to accurately assess respiratory effort, and recorded a sleep diary to detect disorganized sleep habits and disturbed sleeping-waking rhythms. We noticed that great respiratory effort existed not only during light sleep, but was also continuously observed during deep sleep despite the total disappearance of obstructive apneas. Furthermore, upper airway obstruction caused by adenoid-tonsillar hypertrophy and hypoxemia during sleep in the rapid eye movement stage were found to be closely related to the sudden infant death syndrome. A sleep diary recording sleep habits and regularity of night sleep seems to provide reliable indicators for the determination of surgical intervention.
报告了腺样体 - 扁桃体肥大引起的呼吸障碍的病理生理结果。在每种情况下，我们监测食管内压力以准确评估呼吸努力，并记录睡眠日记以检测睡眠习惯紊乱和睡眠 - 觉醒节律紊乱。我们注意到，不仅在浅睡眠期间存在巨大的呼吸努力，而且在深睡眠期间尽管阻塞性呼吸暂停完全消失，但仍持续观察到这种情况。此外，发现腺样体 - 扁桃体肥大引起的上气道阻塞和快速眼动睡眠期睡眠期间的低氧血症与婴儿猝死综合征密切相关。记录睡眠习惯和夜间睡眠规律的睡眠日记似乎为确定手术干预提供了可靠指标。
