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人血管性血友病因子的亚结构

Substructure of human von Willebrand factor.

作者信息

Fowler W E, Fretto L J, Hamilton K K, Erickson H P, McKee P A

出版信息

J Clin Invest. 1985 Oct;76(4):1491-500. doi: 10.1172/JCI112129.

DOI:10.1172/JCI112129
PMID:2932468
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC424111/
Abstract

Using electron microscopy, we have visualized the substructure of human von Willebrand factor (vWf) purified by two different approaches. vWf multimers, which appear as flexible strands varying in length up to 2 micron, consist of dimeric units (protomers) polymerized linearly in an end-to-end fashion through disulfide bonds. Examination of small multimers (e.g., one-mers, two-mers, and three-mers) suggests that each protomer consists of two large globular end domains (22 X 6.5 nm) connected to a small central node (6.4 X 3.4 nm) by two flexible rod domains each approximately 34 nm long and approximately 2 nm in diameter. The protomer is 120 nm in length when fully extended. These same structural features are seen both in vWf molecules that were rapidly purified from fresh plasma by a new two-step procedure and in those purified from lyophilized intermediate-purity Factor VIII/vWf concentrates. The 240,000-mol wt subunit observed by gel electrophoresis upon complete reduction of vWf apparently contains both a rod domain and a globular domain and corresponds to one half of the protomer. Two subunits are disulfide-linked, probably near their carboxyl termini, to form the protomer; disulfide bonds in the amino-terminal globular ends link promoters to form vWf multimers. The vWf multimer strands have at least two morphologically distinct types of ends, which may result from proteolytic cleavage in the globular domains after formation of large linear polymers. In addition to releasing fragments that were similar in size and shape to the repeating protomeric unit, plasmic degradation of either preparation of vWf reduced the size of multimers, but had no detectable effect on the substructure of internal protomers.

摘要

通过电子显微镜,我们已经观察到了通过两种不同方法纯化的人血管性血友病因子(vWf)的亚结构。vWf多聚体呈现为长度可达2微米的柔性链,由二聚体单元(原体)通过二硫键以端对端的方式线性聚合而成。对小多聚体(例如单体、二聚体和三聚体)的检查表明,每个原体由两个大的球形末端结构域(22×6.5纳米)组成,这两个末端结构域通过两个柔性杆状结构域连接到一个小的中央节点(6.4×3.4纳米),每个杆状结构域长约34纳米,直径约2纳米。原体完全伸展时长度为120纳米。在通过一种新的两步法从新鲜血浆中快速纯化的vWf分子以及从冻干的中纯度因子VIII/vWf浓缩物中纯化的vWf分子中都可以看到这些相同的结构特征。在vWf完全还原后通过凝胶电泳观察到的240,000道尔顿分子量的亚基显然同时包含一个杆状结构域和一个球形结构域,并且相当于原体的一半。两个亚基通过二硫键连接,可能在它们的羧基末端附近,形成原体;氨基末端球形末端的二硫键将原体连接形成vWf多聚体。vWf多聚体链至少有两种形态上不同的末端类型,这可能是由于在形成大型线性聚合物后球形结构域中的蛋白水解切割所致。除了释放出大小和形状与重复的原体单元相似的片段外,两种vWf制剂的血浆降解都减小了多聚体的大小,但对内部原体的亚结构没有可检测到的影响。

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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/71cf/424111/246880f62559/jcinvest00124-0213-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/71cf/424111/ec94a9f8bd32/jcinvest00124-0214-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/71cf/424111/406a452be169/jcinvest00124-0215-a.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/71cf/424111/21122c8165b2/jcinvest00124-0215-f.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/71cf/424111/238ba9a0e8a3/jcinvest00124-0216-a.jpg
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