《Tspan8和Tspan8/CD151双敲除小鼠中白细胞迁移、血管生成、伤口修复和转移的畸变表明Tspan8和CD151具有互补活性》的勘误 [《生物化学与生物物理学学报》1865(2) (2018) 379 - 391] - Suppr

Corrigendum to "Distorted leukocyte migration, angiogenesis, wound repair and metastasis in Tspan8 and Tspan8/CD151 double knockout mice indicate complementary activities of Tspan8 and CD51" [Biochim. Biophys. Acta 1865(2) (2018) 379-391].

作者信息

Zhao Kun, Erb Ulrike, Hackert Thilo, Zöller Margot, Yue Shijing

机构信息

Tumor Cell Biology, University Hospital of Surgery, Heidelberg, Germany; Pancreas Section, University Hospital of Surgery, Heidelberg, Germany.

出版信息

Biochim Biophys Acta Mol Cell Res. 2018 Apr;1865(4):674. doi: 10.1016/j.bbamcr.2018.01.001. Epub 2018 Jan 30.

DOI:10.1016/j.bbamcr.2018.01.001

PMID:29366556

Abstract

摘要

相似文献

Corrigendum to "Distorted leukocyte migration, angiogenesis, wound repair and metastasis in Tspan8 and Tspan8/CD151 double knockout mice indicate complementary activities of Tspan8 and CD51" [Biochim. Biophys. Acta 1865(2) (2018) 379-391].《Tspan8和Tspan8/CD151双敲除小鼠中白细胞迁移、血管生成、伤口修复和转移的畸变表明Tspan8和CD151具有互补活性》的勘误 [《生物化学与生物物理学学报》1865(2) (2018) 379 - 391]

Biochim Biophys Acta Mol Cell Res. 2018 Apr;1865(4):674. doi: 10.1016/j.bbamcr.2018.01.001. Epub 2018 Jan 30.

Distorted leukocyte migration, angiogenesis, wound repair and metastasis in Tspan8 and Tspan8/CD151 double knockout mice indicate complementary activities of Tspan8 and CD51.Tspan8 和 Tspan8/CD151 双基因敲除小鼠中白细胞迁移、血管生成、伤口修复和转移的扭曲表明 Tspan8 和 CD51 具有互补的活性。

Biochim Biophys Acta Mol Cell Res. 2018 Feb;1865(2):379-391. doi: 10.1016/j.bbamcr.2017.11.007. Epub 2017 Nov 11.

Tspan8 and Tspan8/CD151 knockout mice unravel the contribution of tumor and host exosomes to tumor progression.Tspan8 和 Tspan8/CD151 敲除小鼠揭示了肿瘤和宿主外泌体对肿瘤进展的贡献。

J Exp Clin Cancer Res. 2018 Dec 12;37(1):312. doi: 10.1186/s13046-018-0961-6.

Tspan8 and CD151 promote metastasis by distinct mechanisms.Tspan8 和 CD151 通过不同的机制促进转移。

Eur J Cancer. 2013 Sep;49(13):2934-48. doi: 10.1016/j.ejca.2013.03.032. Epub 2013 May 14.

The tetraspanins CD151 and Tspan8 are essential exosome components for the crosstalk between cancer initiating cells and their surrounding.四跨膜蛋白CD151和Tspan8是癌症起始细胞与其周围环境之间相互作用的重要外泌体成分。

Oncotarget. 2015 Feb 10;6(4):2366-84. doi: 10.18632/oncotarget.2958.

Joint features and complementarities of Tspan8 and CD151 revealed in knockdown and knockout models.敲低和敲除模型揭示的Tspan8和CD151的联合特征及互补性。

Biochem Soc Trans. 2017 Apr 15;45(2):437-447. doi: 10.1042/BST20160298.

Activation-induced internalization differs for the tetraspanins CD9 and Tspan8: Impact on tumor cell motility.四跨膜蛋白 CD9 和 Tspan8 的激活诱导内化不同：对肿瘤细胞迁移的影响。

Int J Biochem Cell Biol. 2011 Jan;43(1):106-19. doi: 10.1016/j.biocel.2010.10.002. Epub 2010 Oct 16.

TSPAN8 as a Novel Emerging Therapeutic Target in Cancer for Monoclonal Antibody Therapy.TSPAN8 作为癌症中单克隆抗体治疗的新型新兴治疗靶点。

Biomolecules. 2020 Mar 3;10(3):388. doi: 10.3390/biom10030388.

Corrigendum to "Double knockout of Akt2 and AMPK predisposes cardiac aging without affecting lifespan: Role of autophagy and mitophagy" [Biochim. Biophys. Acta Mol. Basis Dis. 2019 Jul 1; 1865(7): 1865-1875. doi: 10.1016/j.bbadis.2018.08.011].《Akt2和AMPK双敲除易引发心脏衰老但不影响寿命：自噬和线粒体自噬的作用》勘误 [《生物化学与生物物理学报：分子疾病基础》2019年7月1日；1865(7): 1865 - 1875。doi: 10.1016/j.bbadis.2018.08.011]

Biochim Biophys Acta Mol Basis Dis. 2024 Jun;1870(5):167109. doi: 10.1016/j.bbadis.2024.167109. Epub 2024 Mar 13.

Corrigendum to: "Double knockout of Akt2 and AMPK accentuates high fat diet-induced cardiac anomalies through a cGAS-STING-mediated mechanism" [Biochim Biophys Acta Mol. Basis Dis. 1866 2020; (10): 165855. PMID: 32512189].

Biochim Biophys Acta Mol Basis Dis. 2022 Oct 1;1868(10):166457. doi: 10.1016/j.bbadis.2022.166457. Epub 2022 Jun 17.

引用本文的文献

The Tetraspanin Tspan8 Associates with Endothelin Converting Enzyme ECE1 and Regulates Its Activity.四跨膜蛋白Tspan8与内皮素转化酶ECE1相关联并调节其活性。

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Corrigendum to "Distorted leukocyte migration, angiogenesis, wound repair and metastasis in Tspan8 and Tspan8/CD151 double knockout mice indicate complementary activities of Tspan8 and CD51" [Biochim. Biophys. Acta 1865(2) (2018) 379-391].

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