青霉素和噬菌体φX174基因E产物对大肠杆菌自溶的差异诱导作用。
Differential induction of Escherichia coli autolysis by penicillin and the bacteriophage phi X174 gene E product.
作者信息
Halfmann G, Lubitz W
出版信息
J Bacteriol. 1986 May;166(2):683-5. doi: 10.1128/jb.166.2.683-685.1986.
Abstract
The behavior of the temperature-sensitive, penicillin-tolerant Escherichia coli mutant VC44 to endogenously induced autolysis by the bacteriophage phi X174 gene E product (gpE) was investigated. Expression of the cloned phi X174 lysis gene showed that cultures of strain VC44 grown at the restricted temperature were fully sensitive to endogenously induced autolysis. The results revealed that the modes of E. coli lysis induction by gpE and by penicillin differ and that the trigger mechanisms for autolysis depend greatly on the specific inducer used.
摘要
研究了温度敏感、耐青霉素的大肠杆菌突变体VC44对噬菌体φX174基因E产物（gpE）内源性诱导自溶的行为。克隆的φX174裂解基因的表达表明，在限制温度下生长的VC44菌株培养物对内源性诱导的自溶完全敏感。结果表明，gpE和青霉素诱导大肠杆菌裂解的方式不同，自溶的触发机制很大程度上取决于所使用的特定诱导剂。
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