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The Cellular Origin of Activated Fibroblasts in the Infarcted and Remodeling Myocardium.

作者信息

Alex Linda, Frangogiannis Nikolaos G

机构信息

From the Department of Medicine (Cardiology), The Wilf Family Cardiovascular Research Institute, Albert Einstein College of Medicine, Bronx, NY.

出版信息

Circ Res. 2018 Feb 16;122(4):540-542. doi: 10.1161/CIRCRESAHA.118.312654.

DOI:10.1161/CIRCRESAHA.118.312654
PMID:29449358
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5820780/
Abstract
摘要

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本文引用的文献

1
Infarct Fibroblasts Do Not Derive From Bone Marrow Lineages.梗死成纤维细胞并非源自骨髓谱系。
Circ Res. 2018 Feb 16;122(4):583-590. doi: 10.1161/CIRCRESAHA.117.311490. Epub 2017 Dec 21.
2
Opposing Actions of Fibroblast and Cardiomyocyte Smad3 Signaling in the Infarcted Myocardium.成纤维细胞和心肌细胞 Smad3 信号在梗死心肌中的拮抗作用。
Circulation. 2018 Feb 13;137(7):707-724. doi: 10.1161/CIRCULATIONAHA.117.029622. Epub 2017 Dec 11.
3
Genetic lineage tracing defines myofibroblast origin and function in the injured heart.遗传谱系追踪定义了心肌梗死后损伤心脏中肌成纤维细胞的起源和功能。
Nat Commun. 2016 Jul 22;7:12260. doi: 10.1038/ncomms12260.
4
Interacting resident epicardium-derived fibroblasts and recruited bone marrow cells form myocardial infarction scar.心肌梗死后瘢痕由相互作用的心肌内源性成纤维细胞和募集的骨髓细胞形成。
J Am Coll Cardiol. 2015 May 19;65(19):2057-66. doi: 10.1016/j.jacc.2015.03.520.
5
Perivascular Gli1+ progenitors are key contributors to injury-induced organ fibrosis.血管周围Gli1+祖细胞是损伤诱导器官纤维化的关键促成因素。
Cell Stem Cell. 2015 Jan 8;16(1):51-66. doi: 10.1016/j.stem.2014.11.004. Epub 2014 Nov 20.
6
Developmental heterogeneity of cardiac fibroblasts does not predict pathological proliferation and activation.心脏成纤维细胞的发育异质性不能预测病理性增殖和激活。
Circ Res. 2014 Sep 12;115(7):625-35. doi: 10.1161/CIRCRESAHA.115.303794. Epub 2014 Jul 18.
7
Resident fibroblast lineages mediate pressure overload-induced cardiac fibrosis.驻留成纤维细胞谱系介导压力超负荷诱导的心脏纤维化。
J Clin Invest. 2014 Jul;124(7):2921-34. doi: 10.1172/JCI74783. Epub 2014 Jun 17.
8
Cardiac fibrosis in human transplanted hearts is mainly driven by cells of intracardiac origin.人心移植后心脏纤维化主要由心脏内源性细胞驱动。
J Am Coll Cardiol. 2012 Mar 13;59(11):1008-16. doi: 10.1016/j.jacc.2011.11.036.
9
Fibrocytes: emerging effector cells in chronic inflammation.成纤维细胞：慢性炎症中的新兴效应细胞。
Nat Rev Immunol. 2011 Jun;11(6):427-35. doi: 10.1038/nri2990. Epub 2011 May 20.
10
Experimental myocardial infarction triggers canonical Wnt signaling and endothelial-to-mesenchymal transition.实验性心肌梗死引发经典 Wnt 信号通路和血管内皮细胞向间充质细胞转化。
Dis Model Mech. 2011 Jul;4(4):469-83. doi: 10.1242/dmm.006510. Epub 2011 Feb 14.