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Reprogramming Tumor-Immune Cell Interface in Solid and Hematological Malignancies to Enhance Response to Therapy.

作者信息

Di Martino Maria Teresa, Zazzeroni Francesca, Donadelli Massimo, Chiodoni Claudia, Caraglia Michele, Scotlandi Katia, Meschini Stefania, Leonetti Carlo

机构信息

Dept. of Experimental and Clinical Medicine, University of Catanzaro "Magna Graecia", Catanzaro, Italy.

Dept. of Biotechnological and Applied Clinical Sciences, University of L'Aquila, L'Aquila, Italy.

出版信息

J Exp Clin Cancer Res. 2018 Mar 5;37(1):48. doi: 10.1186/s13046-018-0710-x.

DOI:10.1186/s13046-018-0710-x
PMID:29506548
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5838855/
Abstract
摘要

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Blood Adv. 2016 Nov 22;1(1):47-61. doi: 10.1182/bloodadvances.2016000984. eCollection 2016 Nov 29.
2
Innovative Therapy, Monoclonal Antibodies and Beyond.创新疗法、单克隆抗体及其他。
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3
Microenvironmental regulation of tumour angiogenesis.肿瘤血管生成的微环境调控。
Elevated Na/H exchanger 1 (SLC9A1) emerges as a marker for tumorigenesis and prognosis in gliomas.
上调的 Na/H 交换蛋白 1(SLC9A1)成为胶质瘤发生和预后的标志物。
J Exp Clin Cancer Res. 2018 Oct 17;37(1):255. doi: 10.1186/s13046-018-0923-z.
Nat Rev Cancer. 2017 Aug;17(8):457-474. doi: 10.1038/nrc.2017.51. Epub 2017 Jul 14.
4
Metabolic regulation of suppressive myeloid cells in cancer.癌症中抑制性髓系细胞的代谢调控。
Cytokine Growth Factor Rev. 2017 Jun;35:27-35. doi: 10.1016/j.cytogfr.2017.05.002. Epub 2017 May 3.
5
Dual angiopoietin-2 and VEGFA inhibition elicits antitumor immunity that is enhanced by PD-1 checkpoint blockade.双重血管生成素-2 和 VEGFA 抑制引发抗肿瘤免疫,这种免疫可被 PD-1 检查点阻断增强。
Sci Transl Med. 2017 Apr 12;9(385). doi: 10.1126/scitranslmed.aak9670.
6
Diffuse large B-cell lymphoma genotyping on the liquid biopsy.弥漫性大 B 细胞淋巴瘤的液体活检基因分型。
Blood. 2017 Apr 6;129(14):1947-1957. doi: 10.1182/blood-2016-05-719641. Epub 2017 Jan 17.
7
T Cells Redirected to a Minor Histocompatibility Antigen Instruct Intratumoral TNFα Expression and Empower Adoptive Cell Therapy for Solid Tumors.T 细胞重定向至次要组织相容性抗原可指导肿瘤内 TNFα 的表达,并增强过继细胞疗法治疗实体瘤。
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8
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Vaccines (Basel). 2016 Nov 4;4(4):38. doi: 10.3390/vaccines4040038.
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Mesenchymal Transition of High-Grade Breast Carcinomas Depends on Extracellular Matrix Control of Myeloid Suppressor Cell Activity.高级别乳腺癌的间充质转化取决于细胞外基质对髓系抑制细胞活性的调控。
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