评论“滑膜成纤维细胞-中性粒细胞相互作用促进类风湿关节炎中的致病适应性免疫”。
Comment on "Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis".
机构信息
Division of Rheumatology, The Johns Hopkins University School of Medicine, Baltimore, MD 21224, USA.
出版信息
Sci Immunol. 2018 Mar 30;3(21). doi: 10.1126/sciimmunol.aao6234.
Abstract
Discrepancies on the role of citrullination in the induction of experimental arthritis by neutrophils extracellular traps.
摘要
中性粒细胞细胞外诱捕网在诱导实验性关节炎中的瓜氨酸化作用存在差异。
相似文献
1
Comment on "Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis".评论“滑膜成纤维细胞-中性粒细胞相互作用促进类风湿关节炎中的致病适应性免疫”。
Sci Immunol. 2018 Mar 30;3(21). doi: 10.1126/sciimmunol.aao6234.
2
Technical comment on "Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis".关于“滑膜成纤维细胞-中性粒细胞相互作用促进类风湿关节炎中的致病适应性免疫”的技术评论。
Sci Immunol. 2020 Jan 31;5(43). doi: 10.1126/sciimmunol.aax5672.
3
Response to comment on "Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis".对“滑膜成纤维细胞-中性粒细胞相互作用促进类风湿关节炎中的致病适应性免疫”一文的评论的回复。
Sci Immunol. 2018 Mar 30;3(21). doi: 10.1126/sciimmunol.aar3701.
4
Neutrophil extracellular traps as a new paradigm in innate immunity: friend or foe?中性粒细胞胞外陷阱作为固有免疫的新模式：是敌是友？
Periodontol 2000. 2013 Oct;63(1):165-97. doi: 10.1111/prd.12025.
5
Peptidylarginine deiminase 2 is required for tumor necrosis factor alpha-induced citrullination and arthritis, but not neutrophil extracellular trap formation.肿瘤坏死因子α诱导的瓜氨酸化和关节炎需要肽基精氨酸脱亚氨酶2，但中性粒细胞胞外陷阱形成不需要。
J Autoimmun. 2017 Jun;80:39-47. doi: 10.1016/j.jaut.2017.01.006. Epub 2017 Feb 7.
6
IgA Complexes in Plasma and Synovial Fluid of Patients with Rheumatoid Arthritis Induce Neutrophil Extracellular Traps via FcαRI.类风湿关节炎患者血浆和滑液中的IgA复合物通过FcαRI诱导中性粒细胞胞外陷阱。
J Immunol. 2016 Dec 15;197(12):4552-4559. doi: 10.4049/jimmunol.1502353. Epub 2016 Nov 9.
7
Erratum for Research Article "Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis" by C. Carmona-Rivera, P. M. Carlucci, E. Moore, N. Lingampalli, H. Uchtenhagen, E. James, Y. Liu, K. L. Bicker, H. Wahamma, V. Hoffman, A. I. Catrina, P. Thompson, J. H. Buckner, W. H. Robinson, D. A. Fox, M. J. Kaplan.C. 卡尔莫纳 - 里韦拉、P.M. 卡尔卢奇、E. 摩尔、N. 林甘帕利、H. 乌赫滕哈根、E. 詹姆斯、Y. 刘、K.L. 比克、H. 瓦哈马、V. 霍夫曼、A.I. 卡特里娜、P. 汤普森、J.H. 巴克纳、W.H. 罗宾逊、D.A. 福克斯、M.J. 卡普兰所著研究文章《滑膜成纤维细胞与中性粒细胞相互作用促进类风湿性关节炎中的致病性适应性免疫》的勘误
Sci Immunol. 2020 Jan 31;5(43). doi: 10.1126/sciimmunol.aaz9319.
8
Human neutrophils in auto-immunity.自身免疫中的人类中性粒细胞。
Semin Immunol. 2016 Apr;28(2):159-73. doi: 10.1016/j.smim.2016.03.004. Epub 2016 Mar 29.
9
Citrullination of synovial proteins in murine models of rheumatoid arthritis.类风湿性关节炎小鼠模型中滑膜蛋白的瓜氨酸化
Arthritis Rheum. 2003 Sep;48(9):2489-500. doi: 10.1002/art.11229.
10
Neutrophil extracellular traps exacerbate Th1-mediated autoimmune responses in rheumatoid arthritis by promoting DC maturation.中性粒细胞胞外诱捕网通过促进树突状细胞成熟加剧类风湿关节炎中Th1介导的自身免疫反应。
Eur J Immunol. 2016 Nov;46(11):2542-2554. doi: 10.1002/eji.201646542. Epub 2016 Oct 5.
引用本文的文献
1
Contribution of neutrophils in the pathogenesis of rheumatoid arthritis.中性粒细胞在类风湿关节炎发病机制中的作用。
J Biomed Res. 2019 Oct 24;34(2):86-93. doi: 10.7555/JBR.33.20190075.
本文引用的文献
1
Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis.滑膜成纤维细胞与中性粒细胞的相互作用促进类风湿性关节炎中的致病性适应性免疫。
Sci Immunol. 2017 Apr;2(10). doi: 10.1126/sciimmunol.aag3358. Epub 2017 Apr 14.
2
Diverse stimuli engage different neutrophil extracellular trap pathways.多种刺激可激活不同的中性粒细胞胞外诱捕网途径。
Elife. 2017 Jun 2;6:e24437. doi: 10.7554/eLife.24437.
3
Aggregatibacter actinomycetemcomitans-induced hypercitrullination links periodontal infection to autoimmunity in rheumatoid arthritis.伴放线聚集杆菌诱导的高瓜氨酸化将牙周感染与类风湿关节炎中的自身免疫联系起来。
Sci Transl Med. 2016 Dec 14;8(369):369ra176. doi: 10.1126/scitranslmed.aaj1921.
4
A Critical Reappraisal of Neutrophil Extracellular Traps and NETosis Mimics Based on Differential Requirements for Protein Citrullination.基于蛋白质瓜氨酸化的不同需求对中性粒细胞胞外陷阱和NETosis模拟物的批判性重新评估。
Front Immunol. 2016 Nov 4;7:461. doi: 10.3389/fimmu.2016.00461. eCollection 2016.
5
Antibodies to native and citrullinated RA33 (hnRNP A2/B1) challenge citrullination as the inciting principle underlying loss of tolerance in rheumatoid arthritis.针对天然和瓜氨酸化 RA33（hnRNP A2/B1）的抗体挑战了瓜氨酸化作为类风湿关节炎中耐受丧失的根本激发原则。
Ann Rheum Dis. 2016 Nov;75(11):2022-2028. doi: 10.1136/annrheumdis-2015-208529. Epub 2016 Feb 10.
6
Insights into the significance of peptidylarginine deiminase 4 and antibodies against citrullinated antigens in the absence of "true ACPAs" in an experimental model of arthritis: comment on the article by Shelef et al.在关节炎实验模型中，对无“真正抗瓜氨酸化蛋白抗体（ACPA）”情况下肽基精氨酸脱亚氨酶4及抗瓜氨酸化抗原抗体意义的见解：对谢列夫等人文章的评论
Arthritis Rheumatol. 2014 Sep;66(9):2642-4. doi: 10.1002/art.38719.
7
Assessing the citrullinome in rheumatoid arthritis synovial fluid with and without enrichment of citrullinated peptides.评估类风湿关节炎滑液中有无瓜氨酸化肽富集的瓜氨酸组。
J Proteome Res. 2014 Jun 6;13(6):2867-73. doi: 10.1021/pr500030x. Epub 2014 Apr 28.
8
The autoantibody repertoire in periodontitis: a role in the induction of autoimmunity to citrullinated proteins in rheumatoid arthritis? Antibodies against uncitrullinated peptides seem to occur prior to the antibodies to the corresponding citrullinated peptides.牙周炎中的自身抗体库：在类风湿关节炎中诱导针对瓜氨酸化蛋白的自身免疫方面起作用？针对未瓜氨酸化肽段的抗体似乎先于针对相应瓜氨酸化肽段的抗体出现。
Ann Rheum Dis. 2014 Jul;73(7):e46. doi: 10.1136/annrheumdis-2014-205498. Epub 2014 Mar 25.
9
Immune-mediated pore-forming pathways induce cellular hypercitrullination and generate citrullinated autoantigens in rheumatoid arthritis.免疫介导的孔形成途径导致细胞过度瓜氨酸化，并在类风湿关节炎中产生瓜氨酸化自身抗原。
Sci Transl Med. 2013 Oct 30;5(209):209ra150. doi: 10.1126/scitranslmed.3006869.
10
NETs are a source of citrullinated autoantigens and stimulate inflammatory responses in rheumatoid arthritis.NETs 是瓜氨酸化自身抗原的来源，并在类风湿性关节炎中刺激炎症反应。
Sci Transl Med. 2013 Mar 27;5(178):178ra40. doi: 10.1126/scitranslmed.3005580.
Zotero 插件