关于“滑膜成纤维细胞-中性粒细胞相互作用促进类风湿关节炎中的致病适应性免疫”的技术评论。
Technical comment on "Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis".
机构信息
Department of Biochemistry and Molecular Pharmacology, University of Massachusetts Medical School, Worcester, MA 01605, USA.
Department of Chemistry, Boston College, Chestnut Hill, MA 02467, USA.
出版信息
Sci Immunol. 2020 Jan 31;5(43). doi: 10.1126/sciimmunol.aax5672.
Abstract
Reassessment of citrullinome cargo in neutrophil extracellular traps confirms the presence of citrullinated peptides.
摘要
中性粒细胞胞外诱捕网中瓜氨酸化蛋白组的再评估证实了瓜氨酸化肽的存在。
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Erratum for Research Article "Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis" by C. Carmona-Rivera, P. M. Carlucci, E. Moore, N. Lingampalli, H. Uchtenhagen, E. James, Y. Liu, K. L. Bicker, H. Wahamma, V. Hoffman, A. I. Catrina, P. Thompson, J. H. Buckner, W. H. Robinson, D. A. Fox, M. J. Kaplan.C. 卡尔莫纳 - 里韦拉、P.M. 卡尔卢奇、E. 摩尔、N. 林甘帕利、H. 乌赫滕哈根、E. 詹姆斯、Y. 刘、K.L. 比克、H. 瓦哈马、V. 霍夫曼、A.I. 卡特里娜、P. 汤普森、J.H. 巴克纳、W.H. 罗宾逊、D.A. 福克斯、M.J. 卡普兰所著研究文章《滑膜成纤维细胞与中性粒细胞相互作用促进类风湿性关节炎中的致病性适应性免疫》的勘误
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本文引用的文献
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Peptidyl arginine deiminases: detection and functional analysis of protein citrullination.肽基精氨酸脱亚氨酶：蛋白质瓜氨酸化的检测和功能分析。
Curr Opin Struct Biol. 2019 Dec;59:205-215. doi: 10.1016/j.sbi.2019.01.024. Epub 2019 Mar 1.
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Evidence for a direct link between PAD4-mediated citrullination and the oxidative burst in human neutrophils.证据表明 PAD4 介导的瓜氨酸化与人类中性粒细胞中的氧化爆发之间存在直接联系。
Sci Rep. 2018 Oct 15;8(1):15228. doi: 10.1038/s41598-018-33385-z.
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Glutamic acid-valine-citrulline linkers ensure stability and efficacy of antibody-drug conjugates in mice.谷氨酸-缬氨酸-瓜氨酸连接子确保抗体药物偶联物在小鼠体内的稳定性和疗效。
Nat Commun. 2018 Jun 28;9(1):2512. doi: 10.1038/s41467-018-04982-3.
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The Rheumatoid Arthritis-Associated Citrullinome.类风湿关节炎相关瓜氨酸组。
Cell Chem Biol. 2018 Jun 21;25(6):691-704.e6. doi: 10.1016/j.chembiol.2018.03.002. Epub 2018 Apr 5.
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Mining the Human Tissue Proteome for Protein Citrullination.从人类组织蛋白质组中挖掘蛋白瓜氨酸化。
Mol Cell Proteomics. 2018 Jul;17(7):1378-1391. doi: 10.1074/mcp.RA118.000696. Epub 2018 Apr 2.
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7
Potential protein targets of the peptidylarginine deiminase 2 and peptidylarginine deiminase 4 enzymes in rheumatoid synovial tissue and its possible meaning.类风湿性滑膜组织中肽基精氨酸脱亚氨酶2和肽基精氨酸脱亚氨酶4酶的潜在蛋白质靶点及其可能意义。
Eur J Rheumatol. 2016 Jun;3(2):44-49. doi: 10.5152/eurjrheum.2015.0055. Epub 2016 Feb 1.
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Immune-mediated pore-forming pathways induce cellular hypercitrullination and generate citrullinated autoantigens in rheumatoid arthritis.免疫介导的孔形成途径导致细胞过度瓜氨酸化，并在类风湿关节炎中产生瓜氨酸化自身抗原。
Sci Transl Med. 2013 Oct 30;5(209):209ra150. doi: 10.1126/scitranslmed.3006869.
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NETs are a source of citrullinated autoantigens and stimulate inflammatory responses in rheumatoid arthritis.NETs 是瓜氨酸化自身抗原的来源，并在类风湿性关节炎中刺激炎症反应。
Sci Transl Med. 2013 Mar 27;5(178):178ra40. doi: 10.1126/scitranslmed.3005580.
10
Ezrin, radixin and moesin are possible auto-immune antigens in rheumatoid arthritis.埃兹蛋白、根蛋白和膜突蛋白可能是类风湿关节炎中的自身免疫抗原。
Mol Immunol. 1996 Oct;33(15):1171-6. doi: 10.1016/s0161-5890(96)00083-1.
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