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Dlx1/2 和 Otp 协调下丘脑生长激素释放激素和 AgRP 神经元的产生。

Dlx1/2 and Otp coordinate the production of hypothalamic GHRH- and AgRP-neurons.

机构信息

Center for Neuroscience, Korea Institute of Science and Technology (KIST), Seoul, 02792, Korea.

Neuroscience Section, Papé Family Pediatrics Research Center, Department of Pediatrics, Oregon Health and Science University, Portland, OR, 97239, USA.

出版信息

Nat Commun. 2018 May 23;9(1):2026. doi: 10.1038/s41467-018-04377-4.

Abstract

Despite critical roles of the hypothalamic arcuate neurons in controlling the growth and energy homeostasis, the gene regulatory network directing their development remains unclear. Here we report that the transcription factors Dlx1/2 and Otp coordinate the balanced generation of the two functionally related neurons in the hypothalamic arcuate nucleus, GHRH-neurons promoting the growth and AgRP-neurons controlling the feeding and energy expenditure. Dlx1/2-deficient mice show a loss-of-GHRH-neurons and an increase of AgRP-neurons, and consistently develop dwarfism and consume less energy. These results indicate that Dlx1/2 are crucial for specifying the GHRH-neuronal identity and, simultaneously, for suppressing AgRP-neuronal fate. We further show that Otp is required for the generation of AgRP-neurons and that Dlx1/2 repress the expression of Otp by directly binding the Otp gene. Together, our study demonstrates that the identity of GHRH- and AgRP-neurons is synchronously specified and segregated by the Dlx1/2-Otp gene regulatory axis.

摘要

尽管下丘脑弓状神经元在控制生长和能量平衡方面起着关键作用,但指导其发育的基因调控网络仍不清楚。在这里,我们报告转录因子 Dlx1/2 和 Otp 协调了下丘脑弓状核中两种功能相关神经元的平衡产生,GHRH 神经元促进生长,AgRP 神经元控制进食和能量消耗。Dlx1/2 缺陷小鼠表现出 GHRH 神经元缺失和 AgRP 神经元增加,并持续出现侏儒症和能量消耗减少。这些结果表明 Dlx1/2 对于指定 GHRH 神经元的身份至关重要,同时也抑制了 AgRP 神经元的命运。我们进一步表明,Otp 对于 AgRP 神经元的产生是必需的,并且 Dlx1/2 通过直接结合 Otp 基因来抑制 Otp 的表达。总之,我们的研究表明,GHRH 和 AgRP 神经元的身份通过 Dlx1/2-Otp 基因调控轴同步指定和分离。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6a0a/5966420/939a7b6fffc1/41467_2018_4377_Fig1_HTML.jpg

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