猿猴病毒40小t抗原诱导的茶碱抗性不是由环磷酸腺苷介导的。
Simian virus 40 small-t antigen-induced theophylline resistance is not mediated by cyclic AMP.
作者信息
Renz C, Rundell K
出版信息
J Virol. 1985 Jun;54(3):876-8. doi: 10.1128/JVI.54.3.876-878.1985.
Abstract
Small-t antigen of simian virus 40 renders CV-1 cells resistant to growth arrest induced by theophylline and other methylxanthines. Elevated levels of cyclic AMP are not involved in growth arrest of CV-1 cells by methylxanthines, and small-t antigen does not alter cyclic AMP levels dramatically after infection.
摘要
猿猴病毒40的小t抗原使CV-1细胞对茶碱和其他甲基黄嘌呤诱导的生长停滞产生抗性。环磷酸腺苷水平的升高与甲基黄嘌呤导致的CV-1细胞生长停滞无关，并且感染后小t抗原不会显著改变环磷酸腺苷水平。
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