Williams David M, Shrikrishnapalasuriyar Natasha, Price David E, Stephens Jeffrey W
Department of Diabetes and Endocrinology, Morriston Hospital, Swansea, UK.
Diabetes Research Unit, College of Medicine, Swansea University, Swansea, UK.
BMJ Case Rep. 2018 Jul 18;2018:bcr-2018-225821. doi: 10.1136/bcr-2018-225821.
We present a case of a 44-year-old man, civil servant, who presented to the emergency department (ED) following an episode of collapse. He was usually fit and well and while at work he had developed a headache with pins and needles over his face and subsequently collapsed. On arrival to ED, he had a Glasgow Coma Scale (GCS) of 4/15. There was no history of note apart from being prescribed citalopram for depression. Laboratory investigations revealed severe hypophosphataemia (phosphate 0.19 mmol/L) and no other electrolyte abnormality. He was commenced on intravenous phosphate and his GCS improved to 15 within 4 hours. Further investigations revealed no cause for his severe hypophosphataemia and repeat bloods in clinic follow-up showed a normal phosphate level.
我们报告一例44岁男性公务员病例，该患者在一次晕倒后被送往急诊科。他平时身体健康，工作时出现头痛并伴有面部针刺感，随后晕倒。到达急诊科时，他的格拉斯哥昏迷量表（GCS）评分为4/15。除因抑郁症服用西酞普兰外，无其他特殊病史。实验室检查显示严重低磷血症（磷酸盐0.19 mmol/L），无其他电解质异常。他开始接受静脉输注磷酸盐治疗，4小时内GCS改善至15分。进一步检查未发现导致其严重低磷血症的原因，门诊随访复查血液显示磷酸盐水平正常。
