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社交焦虑和抑郁中的情绪调节:表达抑制和认知重评的系统评价。

Emotion regulation in social anxiety and depression: a systematic review of expressive suppression and cognitive reappraisal.

机构信息

Temple University, Philadelphia, PA, United States.

Temple University, Philadelphia, PA, United States.

出版信息

Clin Psychol Rev. 2018 Nov;65:17-42. doi: 10.1016/j.cpr.2018.07.004. Epub 2018 Jul 23.

Abstract

Social anxiety disorder (SAD) and major depressive disorder (MDD) are highly comorbid, and together they result in greater functional impairment and a poorer prognosis than either condition alone. Theoretical models implicate impairments in emotion regulation in the development and maintenance of internalizing disorders, yet there has been no systematic comparison of emotion regulation in social anxiety and depression. The current review presents an in-depth examination of the literature on two widely-studied emotion regulation strategies, expressive suppression (ES) and cognitive reappraisal (CR), in SAD and MDD. Our review indicated that SAD is broadly characterized by an overreliance on ES, which is associated with negative social and emotional consequences. SAD is also characterized by ineffective utilization of CR, which inhibits the potential positive emotional benefits of this adaptive emotion regulation strategy. In contrast, MDD is broadly characterized by an underutilization of CR, which may be particularly detrimental in stressful or uncontrollable situations. For both SAD and MDD, treatment intervention appears to address deficits in CR but not ES. After reviewing the literature, we propose multiple pathways by which impairments in ES and CR may increase risk for the co-occurrence of SAD and MDD. Clinical implications and future research directions are also discussed.

摘要

社交焦虑障碍(SAD)和重性抑郁障碍(MDD)高度共病,它们共同导致的功能损害和预后比任何一种疾病单独存在时都要差。理论模型表明,情绪调节受损是内化障碍的发展和维持的原因,但尚未对社交焦虑和抑郁中的情绪调节进行系统比较。目前的综述对 SAD 和 MDD 中两种广泛研究的情绪调节策略——表达抑制(ES)和认知重评(CR)的文献进行了深入的考察。我们的综述表明,SAD 的特征是广泛依赖 ES,这与负面的社会和情绪后果有关。SAD 还表现出对 CR 的利用不足,这抑制了这种适应性情绪调节策略的潜在积极情绪益处。相比之下,MDD 的特征是广泛缺乏 CR 的利用,这在压力或不可控的情况下可能特别有害。对于 SAD 和 MDD 来说,治疗干预似乎都能解决 CR 的不足,但不能解决 ES 的不足。在回顾文献后,我们提出了多种途径,通过这些途径,ES 和 CR 的损伤可能会增加 SAD 和 MDD 共病的风险。还讨论了临床意义和未来的研究方向。

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