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JAK2 double minutes with resultant simultaneous amplification of JAK2 and CD274 in a therapy-related myelodysplastic syndrome evolving into an acute myeloid leukaemia.

作者信息

Wang Huan-You, Dell'Aquila Marie L, Dvanajscak Zeljko, Bejar Rafael, Broome H Elizabeth, Hsi Eric, Murray Sarah S, Thorson John A

机构信息

Division of Laboratory and Genomic Medicine, Department of Pathology, University of California San Diego, La Jolla, CA, USA.

Division of Medical Genetics, Department of Medicine, University of California San Diego, La Jolla, CA, USA.

出版信息

Br J Haematol. 2019 May;185(3):566-570. doi: 10.1111/bjh.15538. Epub 2018 Aug 21.

DOI:10.1111/bjh.15538
PMID:30132795
Abstract
摘要

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JAK2 double minutes with resultant simultaneous amplification of JAK2 and CD274 in a therapy-related myelodysplastic syndrome evolving into an acute myeloid leukaemia.在一例演变为急性髓系白血病的治疗相关骨髓增生异常综合征中，出现携带JAK2双微体，导致JAK2和CD274同时扩增。
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Acute myeloid leukemias with ring sideroblasts show a unique molecular signature straddling secondary acute myeloid leukemia and acute myeloid leukemia.伴有环形铁粒幼细胞的急性髓系白血病表现出一种独特的分子特征，跨越继发性急性髓系白血病和急性髓系白血病。
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Myelodysplastic syndrome with concomitant del(5q) and JAK2 V617F mutation transformed to acute myeloid leukemia with an additional chromosomal abnormality after a long-term treatment with lenalidomide.伴有del(5q)和JAK2 V617F突变的骨髓增生异常综合征在长期使用来那度胺治疗后转化为急性髓系白血病，并伴有额外的染色体异常。
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