Yazaki Kyoichiro, Enta Kenji, Kataoka Shohei, Kahata Mitsuru, Kumagai Asako, Inoue Koji, Koganei Hiroshi, Otsuka Masato, Ishii Yasuhiro
Department of Cardiology, Cardiovascular Center, Ogikubo Hospital, Tokyo, Japan.
J Cardiol Cases. 2016 Nov 22;15(3):80-83. doi: 10.1016/j.jccase.2016.10.014. eCollection 2017 Mar.
Slow-fast atrioventricular nodal tachycardia (AVNRT) has various electrophysiological aspects due to atrioventricular (AV) nodal physiology. In addition, concomitantly another form of arrhythmia with AVNRT, especially atrial tachycardia (AT), was an infrequent arrhythmia. A 38-year-old female with narrow QRS tachycardia underwent electrophysiological study due to frequent faintness. The electrophysiological study disclosed the coexistence of AT originating from coronary sinus (CS) with slow-fast AVNRT. We easily diagnosed AT originating from CS and terminated with several radiofrequency ablations (RFA) around CS. The diagnosis of slow-fast AVNRT, however, was somewhat difficult due to the following findings: (1) small amount of adenosine triphosphate (ATP) could terminate slow-fast AVNRT reproducibly; (2) we could provoke slow-fast AVNRT only by RV pacing with isoproterenol infusion. With other electrophysiological findings, we diagnosed slow-fast AVNRT. Radiofrequency energy was delivered initially in the posteroseptal region, followed by inside CS, and finally in the middle septal region, which completed the slow pathway ablation. After the procedure, we could never provoke these arrhythmias. < Coexistence of focal AT originating from CS with slow-fast AVNRT is a rare phenomenon. Furthermore, slow-fast AVNRT could show unusual characteristic as following: (1) small amount of ATP terminates slow-fast AVNRT; (2) atrial pacing never provoked slow-fast AVNRT with isoproterenol infusion whereas ventricular pacing did, which depends on the physiological characteristic of the dual AV nodal pathway. Accordingly, we should precisely assess the obtained electrophysiological findings.>.
由于房室(AV)结的生理特性,慢-快型房室结折返性心动过速(AVNRT)具有多种电生理特征。此外,AVNRT常合并另一种心律失常,尤其是房性心动过速(AT),但这种情况并不常见。一名38岁女性因频繁晕厥接受了窄QRS波心动过速的电生理检查。电生理检查发现起源于冠状窦(CS)的AT与慢-快型AVNRT共存。我们很容易诊断出起源于CS的AT,并通过在CS周围进行几次射频消融(RFA)将其终止。然而,由于以下发现,慢-快型AVNRT的诊断有些困难:(1)少量三磷酸腺苷(ATP)可重复性地终止慢-快型AVNRT;(2)仅在异丙肾上腺素输注下进行右心室起搏才能诱发慢-快型AVNRT。结合其他电生理检查结果,我们诊断为慢-快型AVNRT。首先在房室结后间隔区域释放射频能量,接着在CS内部,最后在中间隔区域,完成了慢径路消融。术后,我们再也无法诱发这些心律失常。<起源于CS的局灶性AT与慢-快型AVNRT共存是一种罕见现象。此外,慢-快型AVNRT可能表现出如下异常特征:(1)少量ATP可终止慢-快型AVNRT;(2)在异丙肾上腺素输注下,心房起搏从未诱发慢-快型AVNRT,而心室起搏却能诱发,这取决于房室结双径路的生理特性。因此,我们应准确评估所获得的电生理检查结果。>