犬红细胞中钠和质子运动偶联的进一步证据。
Further evidence for coupling of sodium and proton movements in dog red blood cells.
作者信息
Funder J, Parker J C, Wieth J O
出版信息
Biochim Biophys Acta. 1987 May 29;899(2):311-2. doi: 10.1016/0005-2736(87)90414-7.
PMID:3034330
Abstract
Using 4,4'-diisothiocyanostilbene-2,2'-disulfonate (DIDS) and tributyltin the sodium transport pathway activated by shrinkage in dog red blood cells is shown to behave as expected for an electroneutral Na+/H+ exchanger. When the driving forces for sodium and protons are equal, flow through the pathway stops. Amiloride inhibits the shrinkage-induced Na+/H+ exchange.
摘要
使用4,4'-二异硫氰基芪-2,2'-二磺酸盐（DIDS）和三丁基锡，发现犬红细胞中因细胞皱缩激活的钠转运途径表现出如预期的电中性Na⁺/H⁺交换体的行为。当钠和质子的驱动力相等时，通过该途径的流动停止。氨氯地平抑制皱缩诱导的Na⁺/H⁺交换。
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