Kim S W, Kim R C, Choi B H, Gordon S K
Spinal Cord Injury Service, Veterans Administration Medical Center, Long Beach, California.
Paraplegia. 1988 Aug;26(4):262-72. doi: 10.1038/sc.1988.40.
The causes of ischaemic myelopathy are described in 25 patients. Nine developed following surgical manipulation or traumatic laceration of the aorta, 1 following intercostal artery ligation, 3 following aortic aneurysm dissection, 2 following myocardial infarction and/or cardiac arrest, 7 in the absence of any specifically identifiable predisposing factors, and 3 in association with decompression sickness. The degree of clinical recovery was greater among those with incomplete spastic (as opposed to complete flaccid) paralysis and among those in whom sensory loss below the level of injury was incomplete. Despite the diversity of mechanisms that may lead to the development of spinal cord ischaemia, structural damage seems in most instances to affect either grey matter or white matter predominantly. Some of the possible reasons for these preferential sites of damage are discussed.
本文描述了25例缺血性脊髓病的病因。9例发生于主动脉手术操作或外伤性撕裂后，1例发生于肋间动脉结扎后，3例发生于主动脉瘤夹层后，2例发生于心肌梗死和/或心脏骤停后，7例无任何明确可识别的易感因素，3例与减压病有关。不完全性痉挛性（与完全性弛缓性相对）瘫痪患者以及损伤平面以下感觉丧失不完全的患者临床恢复程度更大。尽管导致脊髓缺血发生的机制多种多样，但在大多数情况下，结构损伤似乎主要影响灰质或白质。本文讨论了这些优先损伤部位的一些可能原因。
