Reagan T J, Trautmann J C
Arch Neurol. 1978 Mar;35(3):133-7. doi: 10.1001/archneur.1978.00500270015004.
A 65-year-old man suffered a midbrain infarct after coronary artery bypass surgery. He was left with a severe neuro-ophthalmologic deficit consisting of paralysis of upward and downward vertical gaze, weakness of adduction of the left eye, a dilated fixed left pupil, and partial right Horner's syndrome. He died 31 months after the episode. Postmortem examination disclosed an infarct involving parts of both oculomotor nuclei as well as supranuclear structures thought to be involved in the mediation of vertical eye movements.
一名65岁男性在冠状动脉搭桥手术后发生中脑梗死。他遗留有严重的神经眼科缺陷，包括向上和向下垂直凝视麻痹、左眼内收无力、左侧瞳孔散大固定以及部分右侧霍纳综合征。发病31个月后他死亡。尸检发现梗死累及动眼神经核的部分区域以及被认为参与垂直眼球运动调节的核上结构。
