• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

勘误：早期生长反应基因-2通过调节转录因子CEBPβ对M1和M2巨噬细胞的激活及可塑性至关重要。

Corrigendum: Early Growth Response Gene-2 Is Essential for M1 and M2 Macrophage Activation and Plasticity by Modulation of the Transcription Factor CEBPβ.

作者信息

Veremeyko Tatyana, Yung Amanda W Y, Anthony Daniel C, Strekalova Tatyana, Ponomarev Eugene D

机构信息

Faculty of Medicine, School of Biomedical Sciences, The Chinese University of Hong Kong, Shatin, Hong Kong.

Department of Pharmacology, University of Oxford, Oxford, United Kingdom.

出版信息

Front Immunol. 2018 Dec 13;9:2923. doi: 10.3389/fimmu.2018.02923. eCollection 2018.

DOI:10.3389/fimmu.2018.02923
PMID:30588236
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6301196/
Abstract

[This corrects the article DOI: 10.3389/fimmu.2018.02515.].

摘要

[本文更正了文章的数字对象标识符：10.3389/fimmu.2018.02515。]

相似文献

1
Corrigendum: Early Growth Response Gene-2 Is Essential for M1 and M2 Macrophage Activation and Plasticity by Modulation of the Transcription Factor CEBPβ.勘误：早期生长反应基因-2通过调节转录因子CEBPβ对M1和M2巨噬细胞的激活及可塑性至关重要。
Front Immunol. 2018 Dec 13;9:2923. doi: 10.3389/fimmu.2018.02923. eCollection 2018.
2
Early Growth Response Gene-2 Is Essential for M1 and M2 Macrophage Activation and Plasticity by Modulation of the Transcription Factor CEBPβ.早期生长反应基因-2 通过调节转录因子 C/EBPβ 对 M1 和 M2 巨噬细胞的激活和可塑性至关重要。
Front Immunol. 2018 Nov 1;9:2515. doi: 10.3389/fimmu.2018.02515. eCollection 2018.
3
Corrigendum: Macrophage Polarization: Different Gene Signatures in M1(LPS+) vs. Classically and M2(LPS-) vs. Alternatively Activated Macrophages.勘误：巨噬细胞极化：M1（LPS+）与经典激活巨噬细胞以及M2（LPS-）与替代激活巨噬细胞的不同基因特征
Front Immunol. 2020 Feb 25;11:234. doi: 10.3389/fimmu.2020.00234. eCollection 2020.
4
TLR2 stimulation impairs anti-inflammatory activity of M2-like macrophages, generating a chimeric M1/M2 phenotype.TLR2 刺激会损害 M2 样巨噬细胞的抗炎活性，从而产生 M1/M2 表型的嵌合体。
Arthritis Res Ther. 2017 Nov 2;19(1):245. doi: 10.1186/s13075-017-1447-1.
5
GWAS-identified CCR1 and IL10 loci contribute to M1 macrophage-predominant inflammation in Behçet's disease.GWAS 鉴定的 CCR1 和 IL10 基因座有助于贝赫切特病中 M1 巨噬细胞占优势的炎症反应。
Arthritis Res Ther. 2018 Jun 12;20(1):124. doi: 10.1186/s13075-018-1613-0.
6
MiRNA-Mediated Macrophage Polarization and its Potential Role in the Regulation of Inflammatory Response.微小RNA介导的巨噬细胞极化及其在炎症反应调节中的潜在作用。
Shock. 2016 Aug;46(2):122-31. doi: 10.1097/SHK.0000000000000604.
7
Corrigendum: Sulforaphane diminishes moonlighting of pyruvate kinase M2 and interleukin 1β expression in M1 (LPS) macrophages.勘误：萝卜硫素减少M1（脂多糖）巨噬细胞中丙酮酸激酶M2的兼职功能及白细胞介素1β的表达。
Front Immunol. 2024 Apr 22;15:1395642. doi: 10.3389/fimmu.2024.1395642. eCollection 2024.
8
Molecular mechanisms that influence the macrophage m1-m2 polarization balance.影响巨噬细胞M1-M2极化平衡的分子机制。
Front Immunol. 2014 Nov 28;5:614. doi: 10.3389/fimmu.2014.00614. eCollection 2014.
9
Notch signaling triggered via the ligand DLL4 impedes M2 macrophage differentiation and promotes their apoptosis.Notch 信号通路通过配体 DLL4 的触发，抑制 M2 巨噬细胞的分化，并促进其凋亡。
Cell Commun Signal. 2018 Jan 10;16(1):4. doi: 10.1186/s12964-017-0214-x.
10
Regulation of immunophenotype modulation of monocytes-macrophages from M1 into M2 by prostate cancer cell-culture supernatant via transcription factor STAT3.通过转录因子 STAT3，前列腺癌细胞培养上清液调节单核细胞-巨噬细胞从 M1 表型向 M2 表型的免疫表型。
Immunol Lett. 2018 Apr;196:140-148. doi: 10.1016/j.imlet.2018.02.009. Epub 2018 Feb 23.

引用本文的文献

1
Multifaceted regulatory mechanisms of the EGR family in tumours and prospects for therapeutic applications (Review).EGR家族在肿瘤中的多方面调控机制及治疗应用前景（综述）
Int J Mol Med. 2025 Jul;56(1). doi: 10.3892/ijmm.2025.5554. Epub 2025 May 30.
2
A literature review of recent advances in gastric cancer treatment: exploring the cross-talk between targeted therapies.胃癌治疗最新进展的文献综述：探索靶向治疗之间的相互作用
Cancer Cell Int. 2025 Jan 24;25(1):23. doi: 10.1186/s12935-025-03655-8.
3
Signs of Deregulated Gene Expression Are Present in Both CD14 and CD14 PBMC From Non-Obese Men With Family History of T2DM.非肥胖且有 2 型糖尿病家族史的男性的 CD14 和 CD14 PBMC 中均存在基因表达失调的迹象。
Front Endocrinol (Lausanne). 2021 Feb 15;11:582732. doi: 10.3389/fendo.2020.582732. eCollection 2020.