Ali Shihab, Wightman Rachel, Hack Jason
Department of Emergency Medicine, Brown University, 55 Claverick Street, Suite 100, Providence, RI 02903.
Department of Emergency Medicine, and Division of Emergency Toxicology, Brown Emergency Medicine, 55 Claverick Street, Suite 100, Providence, RI.
R I Med J (2013). 2019 Feb 1;102(1):55-57.
Cement is widely used in construction. Acute exposures with immediate sequelae have been infrequently described. This case report describes a man who developed multifocal pneumonitis with acute respiratory distress syndrome (ARDs) and respiratory failure one day after cement dust exposure. Chromium, cobalt, and nickel components in cement may cause pulmonary tissue irritation. Sand and gravel in cement may cause direct abrasive injury. Inhalation may cause direct thermal injury through an exothermic reaction. The silicon dioxide component has been shown to cause pulmonary injury through cytokine-mediated inflammation. Cement batches for smaller-scale construction jobs are often mixed onsite increasing exposure risk. Implementation of personal protective equipment has been shown to reduce respiratory symptoms among cement workers, underscoring the need for occupational health standards and further research. [Full article available at http://rimed.org/rimedicaljournal-2019-02.asp].
水泥在建筑中被广泛使用。关于急性接触后立即出现后遗症的情况鲜有描述。本病例报告描述了一名男子，在接触水泥粉尘一天后出现多灶性肺炎伴急性呼吸窘迫综合征（ARDS）和呼吸衰竭。水泥中的铬、钴和镍成分可能会引起肺组织刺激。水泥中的沙子和砾石可能会造成直接的磨蚀性损伤。吸入可能通过放热反应导致直接热损伤。二氧化硅成分已被证明可通过细胞因子介导的炎症引起肺损伤。小规模建筑工程用的水泥批次通常在现场混合，增加了接触风险。已证明使用个人防护设备可减少水泥工人的呼吸道症状，这突出了职业健康标准和进一步研究的必要性。[全文可在http://rimed.org/rimedicaljournal-2019-02.asp获取]
