Department of Orthopaedic Surgery, University of Utah School of Medicine, Salt Lake City, UT.
Department of Orthopedic Surgery, Harborview Medical Center, University of Washington, Seattle, WA.
J Orthop Trauma. 2019 Mar;33(3):e74-e78. doi: 10.1097/BOT.0000000000001363.
To identify the incidence and fracture characteristics associated with syndesmotic injury in tibial plafond fractures and report the incidence of posttraumatic osteoarthrosis (PTOA).
Retrospective comparative study.
Two level-1 academic trauma centers.
PATIENTS/PARTICIPANTS: Of the 735 tibial plafond fractures (OTA/AO 43-B3 and 43-C) treated from January 2006 through December 2015, 108 patients (108/735, 15%) were identified with syndesmosis injury.
Either acute or missed syndesmotic injury.
PTOA.
Fourteen fractures (14/735, 2%) had missed syndesmotic injury. Volkmann fragment of ≤10 mm (P = 0.04) and fibular avulsion fracture (P = 0.05) were significantly more common in missed syndesmosis. Ninety fractures (14/14 missed, 76/94 acute) had greater than 12-month follow-up (mean, 26 months; range, 12-102 months). Nearly all patients with missed syndesmosis injury developed arthrosis (13/14, 93%), and 45% (34/76 fractures) of plafond fractures with acute syndesmosis injury developed arthrosis (P < 0.001). Although controlling for malreduction, patients with missed syndesmosis had significantly more PTOA development (P = 0.018). Controlling for malreduction, patients with syndesmotic fixation and a ≤10-mm Chaput or Volkmann fragment or fibular avulsion fracture (8/31, 26%) were less likely to develop PTOA than if they had a similar fracture pattern without syndesmotic fixation (9/10, 90%) (P = 0.011).
Fifteen percent of tibia plafond fractures have a syndesmosis or syndesmotic equivalent injury. Missed syndesmosis injury has a high rate of PTOA development. Patients with a ≤10-mm Chaput or Volkmann fragment and/or fibular avulsion fracture benefit from syndesmotic fixation.
Prognostic Level IV. See Instructions for Authors for a complete description of levels of evidence.
确定与胫骨平台骨折相关的下胫腓联合损伤的发生率和骨折特征,并报告创伤后骨关节炎(PTOA)的发生率。
回顾性比较研究。
两个 1 级学术创伤中心。
患者/参与者:2006 年 1 月至 2015 年 12 月,共治疗了 735 例胫骨平台骨折(OTA/AO 43-B3 和 43-C),其中 108 例(108/735,15%)被确定为下胫腓联合损伤。
无论是急性还是漏诊的下胫腓联合损伤。
PTOA。
14 例(735 例中的 14 例,2%)存在漏诊的下胫腓联合损伤。Volkmann 片段小于或等于 10mm(P=0.04)和腓骨撕脱骨折(P=0.05)在下胫腓联合漏诊中更为常见。90 例(14 例漏诊,94 例急性)骨折获得超过 12 个月的随访(平均 26 个月;范围 12-102 个月)。几乎所有漏诊下胫腓联合损伤的患者都发生了关节炎(13/14,93%),76 例(94 例急性)中只有 45%(34 例)发生了平台骨折关节炎(P<0.001)。尽管控制了复位不良,漏诊下胫腓联合损伤的患者发生 PTOA 的几率明显更高(P=0.018)。在控制复位不良的情况下,下胫腓联合固定且Chaput 或 Volkmann 片段≤10mm或腓骨撕脱骨折的患者(31 例中的 8 例,26%)发生 PTOA 的可能性低于未固定且具有类似骨折模式的患者(10 例中的 9 例,90%)(P=0.011)。
15%的胫骨平台骨折存在下胫腓联合或类似损伤。漏诊的下胫腓联合损伤有很高的 PTOA 发展率。Chaput 或 Volkmann 片段小于或等于 10mm 且/或腓骨撕脱骨折的患者,从下胫腓联合固定中获益。
预后 IV 级。有关证据水平的完整描述,请参见作者说明。