Platelet respiration.
作者信息
Kholmukhamedov Andaleb, Jobe Shawn
机构信息
Versiti Blood Research Institute, Milwaukee, WI.
出版信息
Blood Adv. 2019 Feb 26;3(4):599-602. doi: 10.1182/bloodadvances.2018025155.
Abstract
摘要
相似文献
1
Platelet respiration.血小板呼吸
Blood Adv. 2019 Feb 26;3(4):599-602. doi: 10.1182/bloodadvances.2018025155.
2
Dichloroacetate, an inhibitor of pyruvate dehydrogenase kinases, inhibits platelet aggregation and arterial thrombosis.双氯乙酸盐，丙酮酸脱氢酶激酶的抑制剂，可抑制血小板聚集和动脉血栓形成。
Blood Adv. 2018 Aug 14;2(15):2029-2038. doi: 10.1182/bloodadvances.2018022392.
3
Thrombin-induced activation of the novel 33-kDa serine/threonine kinase in human platelets.凝血酶诱导人血小板中新型33 kDa丝氨酸/苏氨酸激酶的激活。
Pol J Pharmacol. 1996 Jan-Feb;48(1):105-7.
4
Variations in platelet protein associated with arterial thrombosis.与动脉血栓形成相关的血小板蛋白变异
Thromb Res. 2008;122(5):640-7. doi: 10.1016/j.thromres.2008.01.017. Epub 2008 Apr 16.
5
Nucleotide-binding oligomerization domain 2 receptor is expressed in platelets and enhances platelet activation and thrombosis.核苷酸结合寡聚化结构域2受体在血小板中表达，并增强血小板活化和血栓形成。
Circulation. 2015 Mar 31;131(13):1160-70. doi: 10.1161/CIRCULATIONAHA.114.013743. Epub 2015 Feb 17.
6
Receptor-interacting protein kinase 3 promotes platelet activation and thrombosis.受体相互作用蛋白激酶3促进血小板活化和血栓形成。
Proc Natl Acad Sci U S A. 2017 Mar 14;114(11):2964-2969. doi: 10.1073/pnas.1610963114. Epub 2017 Feb 27.
7
Class III PI3K Positively Regulates Platelet Activation and Thrombosis via PI(3)P-Directed Function of NADPH Oxidase.III类磷脂酰肌醇-3激酶通过NADPH氧化酶的PI(3)P导向功能正向调节血小板活化和血栓形成。
Arterioscler Thromb Vasc Biol. 2017 Nov;37(11):2075-2086. doi: 10.1161/ATVBAHA.117.309751. Epub 2017 Sep 7.
8
Inhibition of the MEK/ERK pathway has no effect on agonist-induced aggregation of human platelets.抑制MEK/ERK信号通路对激动剂诱导的人血小板聚集没有影响。
Biochem Pharmacol. 2003 Apr 15;65(8):1243-50. doi: 10.1016/s0006-2952(03)00069-8.
9
Monomeric C-reactive protein is prothrombotic and dissociates from circulating pentameric C-reactive protein on adhered activated platelets under flow.单体 C 反应蛋白在流动状态下与附着的活化血小板结合，从而具有促血栓形成作用，并从循环中的五聚体 C 反应蛋白中解离出来。
Cardiovasc Res. 2011 Nov 1;92(2):328-37. doi: 10.1093/cvr/cvr226. Epub 2011 Aug 22.
10
Threonine phosphorylation of integrin beta3 in calyculin A-treated platelets is selectively sensitive to 5'-iodotubercidin.在经花萼海绵诱癌素A处理的血小板中，整合素β3的苏氨酸磷酸化对5'-碘杀结核菌素具有选择性敏感性。
Biochim Biophys Acta. 2007 Feb;1773(2):185-91. doi: 10.1016/j.bbamcr.2006.08.053. Epub 2006 Sep 12.
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Deciphering Platelets: Are They Cells or an Evolved Form of Extracellular Vesicles?解读血小板：它们是细胞还是细胞外囊泡的一种进化形式？
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Modulatory effects of dietary saturated fatty acids on platelet mitochondrial function following short-term exposure to ambient Particulate Matter (PM).膳食饱和脂肪酸对短期暴露于环境颗粒物（PM）后血小板线粒体功能的调节作用。
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Metabolic targeting of platelets to combat thrombosis: dawn of a new paradigm?靶向血小板代谢以防治血栓形成：新范式的曙光？
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4
MTH1 protects platelet mitochondria from oxidative damage and regulates platelet function and thrombosis.MTH1 可保护血小板线粒体免受氧化损伤，并调节血小板功能和血栓形成。
Nat Commun. 2023 Aug 10;14(1):4829. doi: 10.1038/s41467-023-40600-7.
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The Use of Triphenyl Phosphonium Cation Enhances the Mitochondrial Antiplatelet Effect of the Compound Magnolol.三苯基鏻阳离子的使用增强了厚朴酚的线粒体抗血小板作用。
Pharmaceuticals (Basel). 2023 Jan 30;16(2):210. doi: 10.3390/ph16020210.
6
Mitochondrial pyruvate dehydrogenase kinases contribute to platelet function and thrombosis in mice by regulating aerobic glycolysis.线粒体丙酮酸脱氢酶激酶通过调节有氧糖酵解促进小鼠血小板功能和血栓形成。
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7
Isotopically nonstationary C metabolic flux analysis in resting and activated human platelets.静息和激活的人血小板中同位素非稳定态 C 代谢通量分析。
Metab Eng. 2022 Jan;69:313-322. doi: 10.1016/j.ymben.2021.12.007. Epub 2021 Dec 22.
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Megakaryopoiesis and Platelet Biology: Roles of Transcription Factors and Emerging Clinical Implications.巨核细胞生成和血小板生物学：转录因子的作用和新兴的临床意义。
Int J Mol Sci. 2021 Sep 5;22(17):9615. doi: 10.3390/ijms22179615.
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Long-term treated HIV infection is associated with platelet mitochondrial dysfunction.长期治疗的 HIV 感染与血小板线粒体功能障碍有关。
Sci Rep. 2021 Mar 18;11(1):6246. doi: 10.1038/s41598-021-85775-5.
10
Antioxidant prevents clearance of hemostatically competent platelets after long-term cold storage.抗氧化剂可防止止血功能正常的血小板在长期冷藏后被清除。
Transfusion. 2021 Feb;61(2):557-567. doi: 10.1111/trf.16200. Epub 2020 Nov 27.
本文引用的文献
1
Glucose metabolism and metabolic flexibility in blood platelets.血小板中的葡萄糖代谢和代谢灵活性。
J Thromb Haemost. 2018 Nov;16(11):2300-2314. doi: 10.1111/jth.14274. Epub 2018 Sep 27.
2
Dichloroacetate, an inhibitor of pyruvate dehydrogenase kinases, inhibits platelet aggregation and arterial thrombosis.双氯乙酸盐，丙酮酸脱氢酶激酶的抑制剂，可抑制血小板聚集和动脉血栓形成。
Blood Adv. 2018 Aug 14;2(15):2029-2038. doi: 10.1182/bloodadvances.2018022392.
3
Rapid kinetics of changes in oxygen consumption rate in thrombin-stimulated platelets measured by high-resolution respirometry.高分辨率呼吸测量法测量凝血酶刺激血小板耗氧量率的快速变化。
Biochem Biophys Res Commun. 2018 Sep 18;503(4):2721-2727. doi: 10.1016/j.bbrc.2018.08.031. Epub 2018 Aug 7.
4
Early alterations in platelet mitochondrial function are associated with survival and organ failure in patients with septic shock.血小板线粒体功能的早期改变与脓毒性休克患者的生存率及器官功能衰竭相关。
J Crit Care. 2016 Feb;31(1):63-7. doi: 10.1016/j.jcrc.2015.10.005. Epub 2015 Oct 26.
5
Metabolic plasticity in resting and thrombin activated platelets.静息和凝血酶激活血小板中的代谢可塑性。
PLoS One. 2015 Apr 13;10(4):e0123597. doi: 10.1371/journal.pone.0123597. eCollection 2015.
6
Platelet mitochondrial dysfunction in critically ill patients: comparison between sepsis and cardiogenic shock.危重症患者血小板线粒体功能障碍：脓毒症与心源性休克的比较
Crit Care. 2015 Feb 11;19(1):39. doi: 10.1186/s13054-015-0762-7.
7
The contributions of respiration and glycolysis to extracellular acid production.呼吸作用和糖酵解对细胞外产酸的贡献。
Biochim Biophys Acta. 2015 Feb;1847(2):171-181. doi: 10.1016/j.bbabio.2014.10.005. Epub 2014 Oct 27.
8
Platelet bioenergetic screen in sickle cell patients reveals mitochondrial complex V inhibition, which contributes to platelet activation.镰状细胞病患者的血小板生物能量筛选显示线粒体复合物 V 抑制，这有助于血小板活化。
Blood. 2014 May 1;123(18):2864-72. doi: 10.1182/blood-2013-09-529420. Epub 2014 Mar 27.
9
Platelet mitochondrial membrane depolarization reflects disease severity in patients with sepsis and correlates with clinical outcome.血小板线粒体膜去极化反映脓毒症患者的疾病严重程度，并与临床结局相关。
Crit Care. 2014 Feb 12;18(1):R31. doi: 10.1186/cc13724.
10
Aldose reductase-mediated phosphorylation of p53 leads to mitochondrial dysfunction and damage in diabetic platelets.醛糖还原酶介导的p53磷酸化导致糖尿病血小板的线粒体功能障碍和损伤。
Circulation. 2014 Apr 15;129(15):1598-609. doi: 10.1161/CIRCULATIONAHA.113.005224. Epub 2014 Jan 28.
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