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Transfer and distribution of accumulated (14C)polychlorinated biphenyls from maternal to fetal and suckling rats.

作者信息

Takagi Y, Aburada S, Hashimoto K, Kitaura T

出版信息

Arch Environ Contam Toxicol. 1986 Nov;15(6):709-15. doi: 10.1007/BF01054917.

DOI:10.1007/BF01054917
PMID:3098190
Abstract
摘要

相似文献

1
Transfer and distribution of accumulated (14C)polychlorinated biphenyls from maternal to fetal and suckling rats.累积的（14C）多氯联苯从母鼠到胎鼠和乳鼠的转移与分布。
Arch Environ Contam Toxicol. 1986 Nov;15(6):709-15. doi: 10.1007/BF01054917.
2
Studies of the transfer and distribution of [14C]polychlorinated biphenyls from maternal to fetal and suckling rats.[14C]多氯联苯从母体到胎鼠和乳鼠的转移与分布研究。
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3
Transfer of 2,4,5,2',4',5'-hexachlorobiphenyl and 2,2-bis-(p-chlorophenyl),1,1,1-trichloroethane (p,p'-DDT) from maternal to newborn and suckling rats.
Arch Toxicol. 1978 Dec 28;41(3):179-86. doi: 10.1007/BF00354089.
4
Transfer of various polychlorinated biphenyls to the foetuses and offspring of mice.多种多氯联苯向小鼠胎儿及后代的转移。
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5
Transfer of polychlorinated biphenyls to the foetuses and offspring of mice.多氯联苯向小鼠胎儿及后代的转移。
Food Cosmet Toxicol. 1978 Feb;16(1):33-7. doi: 10.1016/s0015-6264(78)80324-1.
6
Transfer and accumulation of persistent organochlorine compounds from bovine dams to newborn and suckling calves.
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Polychlorinated biphenyls: evidence of transplacental passage in the Sherman rat.多氯联苯：在谢尔曼大鼠中经胎盘传递的证据。
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Mobilization of organochlorines from female lipid tissue and transplacental transfer to fetus in a harbour porpoise (Phocoena phocoena) in a contaminated area.
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[Manganese transfer from mothers to fetuses or sucklings during pregnancy and lactation].
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Microglial responses to inflammatory challenge in adult rats altered by developmental exposure to polychlorinated biphenyls in a sex-specific manner.发育过程中接触多氯联苯会以性别特异性方式改变成年大鼠对炎症性挑战的小胶质细胞反应。
Neurotoxicology. 2024 Sep;104:95-115. doi: 10.1016/j.neuro.2024.07.009. Epub 2024 Jul 20.
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Perspective on prenatal polychlorinated biphenyl exposure and the development of the progeny nervous system (Review).产前多氯联苯暴露与子代神经系统发育的研究进展（综述）。
Int J Mol Med. 2021 Aug;48(2). doi: 10.3892/ijmm.2021.4983. Epub 2021 Jun 16.
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Exposure to prenatal PCBs shifts the timing of neurogenesis in the hypothalamus of developing rats.

本文引用的文献

1
General summary and conslusions.一般总结与结论。 你提供的原文中“conslusions”拼写有误，正确的应该是“conclusions” 。
Environ Health Perspect. 1978 Jun;24:191-8. doi: 10.1289/ehp.24-1637211.
2
Occurrence of polychlorinated biphenyls in humans.多氯联苯在人体中的存在情况。
Environ Health Perspect. 1972 Apr;1:73-8. doi: 10.1289/ehp.720173.
3
Pathology of chronic polychlorinated biphenyl (PCB) feeding in rats.
Toxicol Appl Pharmacol. 1984 Sep 15;75(2):278-88. doi: 10.1016/0041-008x(84)90210-2.
产前多氯联苯暴露会改变发育中大鼠下丘脑神经发生的时间。
J Exp Zool A Ecol Integr Physiol. 2020 Oct;333(8):550-560. doi: 10.1002/jez.2404. Epub 2020 Aug 15.
4
Sex-specific effects of developmental exposure to polychlorinated biphenyls on neuroimmune and dopaminergic endpoints in adolescent rats.发育过程中接触多氯联苯对青少年大鼠神经免疫和多巴胺能终点的性别特异性影响。
Neurotoxicol Teratol. 2020 May-Jun;79:106880. doi: 10.1016/j.ntt.2020.106880. Epub 2020 Apr 4.
5
Passing experiences on to future generations: endocrine disruptors and transgenerational inheritance of epimutations in brain and sperm.将经验传递给后代：脑和精子中表观遗传修饰的内分泌干扰物和跨代遗传。
Epigenetics. 2018;13(10-11):1106-1126. doi: 10.1080/15592294.2018.1543506. Epub 2018 Nov 16.
6
Effects of the Endocrine-Disrupting Chemicals, Vinclozolin and Polychlorinated Biphenyls, on Physiological and Sociosexual Phenotypes in F2 Generation Sprague-Dawley Rats.内分泌干扰化学物质（伏氯卓林和多氯联苯）对 F2 代 Sprague-Dawley 大鼠生理和社交行为表型的影响。
Environ Health Perspect. 2018 Sep;126(9):97005. doi: 10.1289/EHP3550.
7
Sex differences in effects of gestational polychlorinated biphenyl exposure on hypothalamic neuroimmune and neuromodulator systems in neonatal rats.妊娠多氯联苯暴露对新生大鼠下丘脑神经免疫和神经调质系统的性别差异影响。
Toxicol Appl Pharmacol. 2018 Aug 15;353:55-66. doi: 10.1016/j.taap.2018.06.002. Epub 2018 Jun 5.
8
Application of a novel social choice paradigm to assess effects of prenatal endocrine-disrupting chemical exposure in rats (Rattus norvegicus).一种新型社会选择范式在评估大鼠（褐家鼠）产前内分泌干扰化学物暴露影响中的应用。
J Comp Psychol. 2018 Aug;132(3):253-267. doi: 10.1037/com0000103. Epub 2018 Apr 23.
9
Perinatal exposure to organohalogen pollutants decreases vasopressin content and its mRNA expression in magnocellular neuroendocrine cells activated by osmotic stress in adult rats.围产期暴露于有机卤素污染物会降低成年大鼠中因渗透压应激而激活的大细胞神经内分泌细胞中血管加压素的含量及其mRNA表达。
Toxicol Appl Pharmacol. 2017 Aug 15;329:173-189. doi: 10.1016/j.taap.2017.05.039. Epub 2017 Jun 1.
10
Anxiety-like behaviors in adulthood are altered in male but not female rats exposed to low dosages of polychlorinated biphenyls in utero.成年雄性大鼠若在子宫内接触低剂量多氯联苯，其焦虑样行为会发生改变，而雌性大鼠则不会。
Horm Behav. 2017 Jan;87:8-15. doi: 10.1016/j.yhbeh.2016.10.011. Epub 2016 Oct 26.
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Polychlorinated biphenyls in the global ecosystem.全球生态系统中的多氯联苯。
Nature. 1968 Dec 14;220(5172):1098-102. doi: 10.1038/2201098a0.
5
Polychlorinated biphenyls: metabolic behavior of pure isomers in pigeons, rats, and brook trout.多氯联苯：纯异构体在鸽子、大鼠和溪鳟中的代谢行为。
Science. 1972 Oct 20;178(4058):312-4. doi: 10.1126/science.178.4058.312.
6
Comparison of polychlorinated biphenyls in yusho patients and ordinary persons.油症患者与普通人中多氯联苯的比较。
Bull Environ Contam Toxicol. 1974 Mar;11(3):213-6. doi: 10.1007/BF01685094.
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Polychlorinated biphenyls: evidence of transplacental passage in the Sherman rat.多氯联苯：在谢尔曼大鼠中经胎盘传递的证据。
Food Cosmet Toxicol. 1973 Jun;11(3):471-6. doi: 10.1016/0015-6264(73)90013-8.
8
Histopathologic studies on liver tumorigenesis induced in mice by technical polychlorinated biphenyls and its promoting effect on liver tumors induced by benzene hexachloride.
J Natl Cancer Inst. 1973 Nov;51(5):1637-46. doi: 10.1093/jnci/51.5.1637.
9
Transfer of polychlorinated biphenyls (PCBs) to newborn infants through the placenta and mothers' milk.多氯联苯（PCBs）通过胎盘和母乳向新生儿的转移。
Arch Environ Contam Toxicol. 1985 Jan;14(1):51-7. doi: 10.1007/BF01055761.
10
Distribution and excretion of (14C)polychlorinated biphenyls after their prolonged administration to male rats.对雄性大鼠长期给予（14C）多氯联苯后的分布与排泄
Toxicol Appl Pharmacol. 1976 Sep;37(3):415-23. doi: 10.1016/0041-008x(76)90203-9.