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用19F核磁共振波谱法在完整雪貂心脏中测量细胞内游离钙浓度。

Intracellular free calcium concentration measured with 19F NMR spectroscopy in intact ferret hearts.

作者信息

Marban E, Kitakaze M, Kusuoka H, Porterfield J K, Yue D T, Chacko V P

出版信息

Proc Natl Acad Sci U S A. 1987 Aug;84(16):6005-9. doi: 10.1073/pnas.84.16.6005.

Abstract

Changes in the intracellular free Ca2+ concentration, [Ca2+]i, mediate excitation-contraction coupling in the heart and contribute to cellular injury during ischemia and reperfusion. To study these processes directly, we measured [Ca2+]i in perfused ferret (Mustela putorius furo) hearts using 19F NMR spectroscopy to detect the 5,5'-difluoro derivative of the Ca2+ chelator 1,2-bis(o-aminophenoxy)ethane-N,N,N',N'-tetraacetic acid (BAPTA). To load cells, hearts were perfused with the acetoxymethyl ester derivative of 5,5'-F2-BAPTA. We measured 19F NMR spectra and left ventricular pressure simultaneously, at rest and during pacing at various external Ca concentrations [( Ca]o). Although contractile force was attenuated by the Ca2+ buffering properties of 5,5'-F2-BAPTA, the decrease in pressure could be overcome by raising [Ca]o. Our mean value of 104 nM for [Ca2+]i at rest in the perfused heart agrees well with previous measurements in isolated ventricular muscle. During pacing at 0.6-4 Hz, time-averaged [Ca2+]i increased; the effect of pacing was augmented by increasing [Ca]o. [Ca2+]i more than tripled during 10-20 min of global ischemia, and returned toward control levels upon reperfusion. This approach promises to be particularly useful in investigating the physiology of intact hearts and the pathophysiology of alterations in the coronary circulation.

摘要

细胞内游离钙离子浓度([Ca2+]i)的变化介导心脏的兴奋-收缩偶联,并在缺血和再灌注期间导致细胞损伤。为了直接研究这些过程,我们使用19F核磁共振波谱法测量灌注雪貂(Mustela putorius furo)心脏中的[Ca2+]i,以检测钙离子螯合剂1,2-双(邻氨基苯氧基)乙烷-N,N,N',N'-四乙酸(BAPTA)的5,5'-二氟衍生物。为了使细胞负载,心脏用5,5'-F2-BAPTA的乙酰氧基甲酯衍生物进行灌注。我们在静息状态和不同外部钙离子浓度([Ca]o)起搏期间同时测量19F核磁共振波谱和左心室压力。尽管5,5'-F2-BAPTA的钙离子缓冲特性减弱了收缩力,但通过提高[Ca]o可以克服压力的降低。我们在灌注心脏静息状态下测得的[Ca2+]i平均值为104 nM,与先前在离体心室肌中的测量结果非常吻合。在0.6 - 4 Hz起搏期间,时间平均[Ca2+]i增加;起搏的效果通过增加[Ca]o而增强。在整体缺血10 - 20分钟期间,[Ca2+]i增加了两倍多,并在再灌注时恢复到对照水平。这种方法有望在研究完整心脏的生理学和冠状动脉循环改变的病理生理学方面特别有用。

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